PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32550901-11	2020	Mechanistically, an ultralow dose of DPI inhibited the production of pro-inflammatory cytokines, (tumor necrosis factor (TNF)-alpha and interleukin (IL)-6), reduced the macrophage infiltration and classical polarization, and induced the ROS generation.	diphenyleneiodonium	37-40	tumor necrosis factor	Mus musculus	98-131	
30196390-8	2018	The ROS scavenger N-acetylcysteine (NAC) and the ROS formation inhibitor diphenyleneiodonium (DPI) also inhibited TNFalpha expression in stimulated macrophages, but unlike 3-AB, NAC and DPI were unable to abolish NFkappaB activity.	diphenyleneiodonium	73-92	tumor necrosis factor	Mus musculus	114-122	
30196390-8	2018	The ROS scavenger N-acetylcysteine (NAC) and the ROS formation inhibitor diphenyleneiodonium (DPI) also inhibited TNFalpha expression in stimulated macrophages, but unlike 3-AB, NAC and DPI were unable to abolish NFkappaB activity.	diphenyleneiodonium	94-97	tumor necrosis factor	Mus musculus	114-122	
18419763-6	2008	Inhibition of reactive oxygen species (ROS) by N-acetyl-cysteine or diphenylene iodonium significantly suppressed the expression of MMP-3, MMP-9, NO and TNF-alpha in LPS-stimulated microglia, suggesting that ROS is an early signaling inducer in LPS-stimulated microglial cells.	diphenyleneiodonium	68-88	tumor necrosis factor	Mus musculus	153-162	
17510837-6	2007	Here, DPI significantly reduced the tumor necrosis factor (TNF)-alpha and MIP-2 responses to quartz, and the MIP-2 response to SPM.	diphenyleneiodonium	6-9	tumor necrosis factor	Mus musculus	36-69	
14576080-8	2004	generation, with diphenylene iodonium suppressed TNF-alpha-induced MCP-1 mRNA accumulation.	diphenyleneiodonium	17-37	tumor necrosis factor	Mus musculus	49-58