PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20630999-9	2010	Treatment with diphenyleneiodonium, a nicotinamide adenine dinucleotide phosphate oxidase (NOX) inhibitor, blocked glyLDL-induced translocation of H-Ras, elevated abundances of HSF1 and PAI-1 in EC, and increased release of hydrogen peroxide from EC.	diphenyleneiodonium	15-34	serpin family E member 1	Homo sapiens	186-191	
24981855-13	2014	Moreover, TGF-beta1 stimulated cell proliferation, migration, collagen I and fibronectin expression, and bFGF and PAI-1 secretion: these effects were attenuated by diphenylene iodonium (DPI), an NADPH oxidase inhibitor, and NOX2 siRNA.	diphenyleneiodonium	164-184	serpin family E member 1	Homo sapiens	114-119	
24981855-13	2014	Moreover, TGF-beta1 stimulated cell proliferation, migration, collagen I and fibronectin expression, and bFGF and PAI-1 secretion: these effects were attenuated by diphenylene iodonium (DPI), an NADPH oxidase inhibitor, and NOX2 siRNA.	diphenyleneiodonium	186-189	serpin family E member 1	Homo sapiens	114-119	
19540572-9	2009	CONCLUSIONS: The NADPH oxidase inhibitors DPI and S 17834 as well as Nox4 silencing decreased PAI-1 synthesis in human cultured endothelial cells demonstrating the involvement of the constitutively active Nox4-containing NADPH oxidase in ROS-mediated PAI-1 transcription via p38 MAPK pathways.	diphenyleneiodonium	42-45	serpin family E member 1	Homo sapiens	94-99	
19401454-4	2009	Diphenyleneiodonium, a NOX inhibitor, prevented the increases of the expression of HSF1 and PAI-1 in EC induced by oxidized LDLs.	diphenyleneiodonium	0-19	serpin family E member 1	Homo sapiens	92-97