PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 11668087-6 2001 ERK1/2 phosphorylation was suppressed by an inhibitor of tyrosine phosphorylation (genestein), cholesterol-binding reagents (filipin or cyclodextrin), or inhibitors of ROS (diphenyleneiodonium, N-acetylcysteine, or catalase), suggesting a role for both membrane cholesterol and ROS in ERK1/2 activation. diphenyleneiodonium 173-192 mitogen-activated protein kinase 3 Bos taurus 0-6