PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17158644-3	2007	ANG II treatment of A10 VSMCs enhanced the production of O(2)(-) and the expression of Nox4 and P47(phox), different subunits of NADPH oxidase, which were attenuated toward control levels by diphenyleneiodonium (DPI).	diphenyleneiodonium	191-210	angiogenin	Homo sapiens	0-3	
17158644-6	2007	In addition, ANG II also enhanced the ERK1/2 phosphorylation that was restored to control levels by DPI.	diphenyleneiodonium	100-103	angiogenin	Homo sapiens	13-16	
17158644-8	2007	In addition, G(s)alpha-mediated diminished stimulation of adenylyl cyclase by stimulatory hormones in ANG II-treated cells was also restored to control levels by DPI.	diphenyleneiodonium	162-165	angiogenin	Homo sapiens	102-105	
17158644-3	2007	ANG II treatment of A10 VSMCs enhanced the production of O(2)(-) and the expression of Nox4 and P47(phox), different subunits of NADPH oxidase, which were attenuated toward control levels by diphenyleneiodonium (DPI).	diphenyleneiodonium	212-215	angiogenin	Homo sapiens	0-3	
16249273-10	2006	LY-294002, a selective phosphatidylinositol (PI 3-kinase) inhibitor, did not prevent ROS accumulation in response to ANG II, whereas DPI blocked ANG II activation of PI 3-kinase, demonstrating that ROS production is upstream of the PI 3-kinase signaling pathway.	diphenyleneiodonium	133-136	angiogenin	Homo sapiens	145-148