PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24666969-0	2014	The involvement of AMPK/GSK3-beta signals in the control of metastasis and proliferation in hepato-carcinoma cells treated with anthocyanins extracted from Korea wild berry Meoru.	Anthocyanins	128-140	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	19-23	
24666969-7	2014	RESULTS: Anthocyanins decrease phospho-GSK3-beta and beta-catenin expression in an in vivo tumor xenograft model, increase AMPK activity in this model, and inhibit cell migration and invasion, possibly by inhibiting MMP-2 (in vitro) and the panendothelial marker, CD31 (in vivo).	Anthocyanins	9-21	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	123-127	
24666969-9	2014	Further, we showed that AMPK siRNA treatment abrogated the ability of anthocyanins to control cell proliferation and metastatic potential, and Compound C, an AMPK inhibitor, could not restore GSK3-beta regulation, as exhibited by anthocyanins in Hep3B cells.	Anthocyanins	70-82	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	24-28	
24666969-10	2014	CONCLUSION: These observations imply that the AMPK-mediated GSK3-beta/beta-catenin circuit plays crucial roles in inhibiting cancer cell proliferation and metastasis in anthocyanin-treated hepato-carcinoma cells of Meoru origin.	Anthocyanins	169-180	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	46-50	
34484410-0	2021	Identification of Stabilization of Malvid Anthocyanins and Antioxidant Stress Activation via the AMPK/SIRT1 Signaling Pathway.	Anthocyanins	42-54	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	97-101	
34484410-14	2021	These results indicate that stabilization malvid anthocyanins exerts an antioxidant activity via the AMPK/SIRT1 signaling pathway.	Anthocyanins	49-61	protein kinase AMP-activated non-catalytic subunit beta 1	Homo sapiens	101-105