PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34418746-11	2021	Moreover, PJ moderated the AlCl3 effects by decreasing the level of thiobarbituric acid reactive substances (TBARS), and enhancing catalase (CAT), superoxide dismutase (SOD), glutathione S-transferase (GST) and glutathione (GSH) activities.	Aluminum Chloride	27-32	hematopoietic prostaglandin D synthase	Mus musculus	175-200	
34418746-11	2021	Moreover, PJ moderated the AlCl3 effects by decreasing the level of thiobarbituric acid reactive substances (TBARS), and enhancing catalase (CAT), superoxide dismutase (SOD), glutathione S-transferase (GST) and glutathione (GSH) activities.	Aluminum Chloride	27-32	hematopoietic prostaglandin D synthase	Mus musculus	202-205	
23315010-7	2013	Administration of AlCl3 developed behavioral deficits, triggered lipid peroxidation (LPO), compromised acetylcholine esterase (AChE) activity, and reduced the levels of superoxide dismutase (SOD), catalase (CAT), glutathione-S-transferase (GST), and reduced glutathione (GSH), and caused histologic aberrations.	Aluminum Chloride	18-23	hematopoietic prostaglandin D synthase	Mus musculus	213-238	
23315010-7	2013	Administration of AlCl3 developed behavioral deficits, triggered lipid peroxidation (LPO), compromised acetylcholine esterase (AChE) activity, and reduced the levels of superoxide dismutase (SOD), catalase (CAT), glutathione-S-transferase (GST), and reduced glutathione (GSH), and caused histologic aberrations.	Aluminum Chloride	18-23	hematopoietic prostaglandin D synthase	Mus musculus	240-243	
23315010-10	2013	AlCl3-induced alterations in the activities of SOD, CAT, GST, AChE and levels of GSH, LPO, activity of AChE, behavioral deficits, histologic aberrations, reactive gliosis, and inflammatory niche were attenuated on treatment with fisetin.	Aluminum Chloride	0-5	hematopoietic prostaglandin D synthase	Mus musculus	57-60