PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 23315010-7 2013 Administration of AlCl3 developed behavioral deficits, triggered lipid peroxidation (LPO), compromised acetylcholine esterase (AChE) activity, and reduced the levels of superoxide dismutase (SOD), catalase (CAT), glutathione-S-transferase (GST), and reduced glutathione (GSH), and caused histologic aberrations. Aluminum Chloride 18-23 hematopoietic prostaglandin D synthase Mus musculus 213-238 23315010-7 2013 Administration of AlCl3 developed behavioral deficits, triggered lipid peroxidation (LPO), compromised acetylcholine esterase (AChE) activity, and reduced the levels of superoxide dismutase (SOD), catalase (CAT), glutathione-S-transferase (GST), and reduced glutathione (GSH), and caused histologic aberrations. Aluminum Chloride 18-23 hematopoietic prostaglandin D synthase Mus musculus 240-243 23315010-10 2013 AlCl3-induced alterations in the activities of SOD, CAT, GST, AChE and levels of GSH, LPO, activity of AChE, behavioral deficits, histologic aberrations, reactive gliosis, and inflammatory niche were attenuated on treatment with fisetin. Aluminum Chloride 0-5 hematopoietic prostaglandin D synthase Mus musculus 57-60 34418746-11 2021 Moreover, PJ moderated the AlCl3 effects by decreasing the level of thiobarbituric acid reactive substances (TBARS), and enhancing catalase (CAT), superoxide dismutase (SOD), glutathione S-transferase (GST) and glutathione (GSH) activities. Aluminum Chloride 27-32 hematopoietic prostaglandin D synthase Mus musculus 175-200 34418746-11 2021 Moreover, PJ moderated the AlCl3 effects by decreasing the level of thiobarbituric acid reactive substances (TBARS), and enhancing catalase (CAT), superoxide dismutase (SOD), glutathione S-transferase (GST) and glutathione (GSH) activities. Aluminum Chloride 27-32 hematopoietic prostaglandin D synthase Mus musculus 202-205