PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8762448-4	1995	Ruthenium red, an inhibitor of Ca(2+)-induced Ca2+ release from intracellular Ca2+ pools, significantly inhibited the release of CGRP.	Ruthenium Red	0-13	calcitonin-related polypeptide alpha	Rattus norvegicus	129-133	
18550765-6	2008	Moreover, the cannabidiol-evoked CGRP release depended on extracellular calcium and was blocked by the nonselective TRP channel blocker, ruthenium red.	Ruthenium Red	137-150	calcitonin-related polypeptide alpha	Rattus norvegicus	33-37	
15249421-5	2004	Capsazepine (vanilloid receptor-1 antagonist; 1-10 microm) and ruthenium red (inhibitor of vanilloid response; 1-30 microm) concentration-dependently inhibited the nicotine-induced vasodilation without affecting the vasodilator response to exogenous CGRP.	Ruthenium Red	63-76	calcitonin-related polypeptide alpha	Rattus norvegicus	250-254	
8891596-5	1996	However, the cardioprotection provided by CGRP- or capsaicin-induced preconditioning was abolished by CGRP-(8-37) and ruthenium red, respectively.	Ruthenium Red	118-131	calcitonin-related polypeptide alpha	Rattus norvegicus	42-46	
8864557-18	1996	The ETX-induced CGRP release is dependent on extracellular Ca2+ influx and involves a ruthenium red-sensitive mechanism.	Ruthenium Red	86-99	calcitonin-related polypeptide alpha	Rattus norvegicus	16-20	
8864557-8	1996	Pretreatment of MAB with capsaicin or ruthenium red inhibited ETX-induced CGRP release by 90% and 71%, respectively.	Ruthenium Red	38-51	calcitonin-related polypeptide alpha	Rattus norvegicus	74-78	
8758688-7	1996	Pretreated MAB with capsaicin or ruthenium red inhibited ETX-induced release of CGRP by 90% and 65% respectively.	Ruthenium Red	33-46	calcitonin-related polypeptide alpha	Rattus norvegicus	80-84	
8758688-10	1996	The release of CGRP is dependent on extra-cellular Ca2+ and Ca2+-induced Ca2+ release from the intracellular Ca2+ store which is sensitive to ruthenium red.	Ruthenium Red	142-155	calcitonin-related polypeptide alpha	Rattus norvegicus	15-19	
8762448-6	1995	The above results suggest that the observed release of CGRP in MAB was mediated by capsaicin-sensitive sensory nerve endings, as a result of Ca(2+)-induced Ca2+ release from the intracellular Ca2+ store which is sensitive to ruthenium red.	Ruthenium Red	225-238	calcitonin-related polypeptide alpha	Rattus norvegicus	55-59	
12506420-6	1995	In contrast, ruthenium red (1 x 10(-5) M), capsaicin antagonist, almost completely prevented acid (pH 4.0)-stimulated CGRP release.	Ruthenium Red	13-26	calcitonin-related polypeptide alpha	Rattus norvegicus	118-122	
1374925-8	1992	Release of CGRP evoked by capsaicin concentrations in the range of 0.1-0.3 microM in either dosage protocol was reduced in the presence of Ruthenium Red (RR, 2.5 microM).	Ruthenium Red	139-152	calcitonin-related polypeptide alpha	Rattus norvegicus	11-15	
1718522-10	1991	The relaxation was partly inhibited by ruthenium red, thus suggesting that capsaicin causes specific release of CGRP from sensory nerve endings in rat coronary arteries.	Ruthenium Red	39-52	calcitonin-related polypeptide alpha	Rattus norvegicus	112-116