PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9025112-6	1996	In addition, 8-OH-DPAT and DOI caused a decrease in plasma CCK levels, whereas the 5-HT1B receptor agonist TFMPP gave rise to an increase in plasma CCK levels.	8-Hydroxy-2-(di-n-propylamino)tetralin	13-22	cholecystokinin	Rattus norvegicus	59-62	
9818982-3	1998	Administration of 8-OH-DPAT, a serotonin1A autoreceptor agonist, reduces the ability of CCK to inhibit feeding.	8-Hydroxy-2-(di-n-propylamino)tetralin	18-27	cholecystokinin	Rattus norvegicus	88-91	
9818982-4	1998	We determined if CCK"s alcohol satiation effect also depends on activity of serotonergic neurons by administering 8-OH-DPAT (120-240 microg/kg) to 23-h water-deprived female and male rats, followed 1 h later by i.p.	8-Hydroxy-2-(di-n-propylamino)tetralin	114-123	cholecystokinin	Rattus norvegicus	17-20	
9818982-6	1998	8-OH-DPAT significantly (p < 0.05) interacted with CCK, and reduced CCK"s ethanol satiation effect when given i.p.	8-Hydroxy-2-(di-n-propylamino)tetralin	0-9	cholecystokinin	Rattus norvegicus	54-57	
9818982-6	1998	8-OH-DPAT significantly (p < 0.05) interacted with CCK, and reduced CCK"s ethanol satiation effect when given i.p.	8-Hydroxy-2-(di-n-propylamino)tetralin	0-9	cholecystokinin	Rattus norvegicus	71-74	
9818982-8	1998	Female rats showed this interaction of 8-OH-DPAT with CCK at a higher dose than males when given i.p., but females were more sensitive to s.c. 8-OH-DPAT"s ability to reduce ethanol intake.	8-Hydroxy-2-(di-n-propylamino)tetralin	39-48	cholecystokinin	Rattus norvegicus	54-57	
8795088-4	1996	8-OH-DPAT also increased insulin and decreased CCK and somatostatin levels, effects that were blocked by pretreatment with the oxytocin antagonist.	8-Hydroxy-2-(di-n-propylamino)tetralin	0-9	cholecystokinin	Rattus norvegicus	47-50	
8795088-5	1996	Taken together, these data suggest that the effect of 8-OH-DPAT on plasma levels of insulin, somatostatin and CCK may be mediated by oxytocin.	8-Hydroxy-2-(di-n-propylamino)tetralin	54-63	cholecystokinin	Rattus norvegicus	110-113	
8795088-9	1996	Since oxytocinergic fibers which originate in the PVN project to the DMX, we suggest that the effect on the release of insulin, CCK and somatostatin induced by the 5 HT1A receptor agonist 8-OH-DPAT may be mediated by an oxytocinergic activation of a vagal mechanism.	8-Hydroxy-2-(di-n-propylamino)tetralin	188-197	cholecystokinin	Rattus norvegicus	128-131	
7619321-4	1995	In a subsequent experiment, this preference was attenuated by coadministration of the 5-hydroxytryptamine1A (5-HT1A) agonist 8-hydroxy-2(di-n-propylamino)tetralin during reexposure, suggesting that CCK interacts with 5-HT to modify incentive value.	8-Hydroxy-2-(di-n-propylamino)tetralin	125-162	cholecystokinin	Rattus norvegicus	198-201