PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30742091-7	2019	Mechanistically, we show that necroptosis upon VNR/IFNbeta/zVAD.fmk treatment is RIP1-independent but relies on IFNbeta-induced gene expression of Z-DNA-binding protein 1 (ZBP1) as shown by quantitative RT-PCR and genetic knockdown experiments.	FMK	64-67	receptor interacting serine/threonine kinase 1	Homo sapiens	81-85	
24030154-7	2013	Importantly, BV6/DAC-induced cell death in the presence of zVAD.fmk is significantly reduced by pharmacological inhibition of key components of necroptosis signaling, that is, receptor-interacting protein (RIP) 1 using necrostatin-1 or mixed lineage kinase domain-like protein (MLKL) using necrosulfonamide.	FMK	64-67	receptor interacting serine/threonine kinase 1	Homo sapiens	176-212