PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19059384-6	2009	Similarly, in ABCG2-overexpressing cells, AG1478 significantly reversed resistance to ABCG2 substrate anticancer drugs and increased intracellular accumulation of [3H]-mitoxantrone as well as fluorescent compound BODIPY-prazosin.	Tritium	164-166	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	14-19	
21935580-10	2012	Drug efflux measurements showed that fumitremorgin C             was able to increase the residual cellular [3H]-etoposide uptake in BCRP-transfected             cells and especially in HEK/R482G ones.	Tritium	109-111	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	133-137	
19059384-7	2009	AG1478 also profoundly inhibited the transport of [3H]-E(2)17betaG and [3H]-methotrexate by ABCG2.	Tritium	51-53	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	92-97	
19059384-7	2009	AG1478 also profoundly inhibited the transport of [3H]-E(2)17betaG and [3H]-methotrexate by ABCG2.	Tritium	51-54	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	92-97	
27780535-7	2016	In transport studies on MDCKII-ABCG2 monolayers etravirine completely abolished the ABCG2-mediated transfer of [3H]-TDF.	Tritium	112-114	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	84-89	
18657189-5	2008	[3H]Daunomycin binding to the ABCG2 R482G isoform was examined in the nucleotide-bound and post-hydrolytic conformations.	Tritium	1-3	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	30-35	
32477943-6	2020	In further tritium-labeled mitoxantrone transportation study, sitravatinib at 3 muM blocked the efflux function mediated by ABCG2 and as a result, increased the intracellular concentration of anticancer drugs.	Tritium	11-18	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	124-129	
30135242-3	2018	The hiPSC-IECs showed the transport activities of P-glycoprotein (P-gp), breast cancer resistance protein (BCRP), and peptide transporter 1 (PEPT1), revealed by using their probe substrates ([3H]digoxin, sulfasalazine, and [14C]glycylsarcosine), and the metabolic activities of CYP3A4, CES2, and CES1, which were clarified using their probe substrates (midazolam, irinotecan, and temocapril).	Tritium	192-194	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	73-105	
16981002-4	2006	EXPERIMENTAL APPROACH: Plasma membranes from insect cells expressing ABCG2 were used to characterise binding of [3H]daunomycin to the multidrug transporter.	Tritium	113-115	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	69-74	
12920197-10	2003	Next, the uptake of 3H-labeled compounds in membrane vesicles from BCRP-transduced K562 (K562/BCRP) cells was investigated.	Tritium	20-22	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	67-71	
12920197-11	2003	3H-labeled estrone sulfate, but not 3H-labeled estrone or 17beta-estradiol, was taken up by membrane vesicles from K562/BCRP cells, and this was ATP-dependent.	Tritium	0-2	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	120-124	
28031414-7	2017	Intracellular concentrations of known P-gp and BCRP substrates [(3H)-paclitaxel and (3H)-prazosin, respectively] were significantly higher (indicating less efflux) in NIH-3T3-MDR1 and MDCK-BCRP cells in the presence of 6-OH-BDE-47, but not BDE-47.	Tritium	64-68	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	47-51	
27300692-5	2016	MDCKII cells overexpressing human BCRP (MDCKII-BCRP) but not those overexpressing human P-gp (MDCKII-MDR cells) had reduced [3H]benzylpenicillin uptake.	Tritium	125-127	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	34-38	
27300692-5	2016	MDCKII cells overexpressing human BCRP (MDCKII-BCRP) but not those overexpressing human P-gp (MDCKII-MDR cells) had reduced [3H]benzylpenicillin uptake.	Tritium	125-127	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	40-51	
27300692-7	2016	While inhibiting BCRP affected [3H]benzylpenicillin cell concentrations it did not affect transepithelial flux in MDCKII-BCRP cells.	Tritium	32-34	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	17-21	
24903205-5	2014	Moreover, WHI-P154 produced a significant increase in the intracellular accumulation of [3H]-mitoxantrone in ABCG2-overexpressing cells.	Tritium	89-91	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	109-114	
22614107-7	2012	Studies on [(3)H]-mitoxantrone accumulation and efflux have shown that zafirlukast increases the intracellular accumulation of [(3)H]-mitoxantrone by directly inhibiting ABCG2-mediated drug efflux.	Tritium	11-16	ATP binding cassette subfamily G member 2 (Junior blood group)	Homo sapiens	170-175