PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28842010-3	2017	Exogenous compounds could compete with the fragment (ED-ES) of genetically engineered beta-galactosidase enzyme (beta-gal) for the binding to ERalpha or beta, thus quantitatively altering the formation of enzymatically active beta-gal and the hydrolysis of luminescent substrate.	beta-D-galactose	86-94	estrogen receptor 1	Homo sapiens	142-149