PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29991461-0	2018	Down-regulation of magnesium transporting molecule, claudin-16, as a possible cause of hypermagnesiuria with the development of tubulo-interstitial nephropathy.	Magnesium	19-28	claudin 16	Rattus norvegicus	52-62	
29991461-7	2018	The gene expression of claudin-16 was decreased in the late phase of UUO (100.2 +- 2.9% at day-0, 90.3 +- 6.3% at day-1, 36.4 +- 1.6% at day-7) which was consistent with the increased urine magnesium excretion.	Magnesium	190-199	claudin 16	Rattus norvegicus	23-33	
29991461-13	2018	Our findings may indicate that the characteristic hypermagnesiuria in TIN is principally caused by the dysfunction of magnesium reabsorption in the thick ascending limb of Henle resulting from a significant decrease in the claudin-16 expression.	Magnesium	118-127	claudin 16	Rattus norvegicus	223-233