PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
17984289-2	2008	In this study, we have explored the role of IL-18, a cytokine with structural similarities to IL-1 beta, in murine LC migration and contact hypersensitivity (CHS), which to oxazolone (OX) and 2-4,dinitrofluorobenzene (DNFB) was suppressed significantly in IL-18 knockout (IL-18-/-) mice and could be rescued by local intradermal administration of IL-18 prior to sensitization, suggesting that the defect in these mice was in the afferent phase of CHS.	Oxazolone	173-182	interleukin 18	Mus musculus	44-49	
27966619-6	2016	Oxazolone treatment stimulated maturation of pro-caspase-1 and pro-IL-1beta, while it reduced IL-18 expression.	Oxazolone	0-9	interleukin 18	Mus musculus	94-99	
27966619-9	2016	Compared to wild-type mice, NLRP3-/- mice exhibited higher sensitivity to oxazolone treatment with enhancement of Th2 cytokine expression and reduction of mature IL-1beta and IL-18 production; this phenotype was rescued by exogenous IL-1beta or IL-18.	Oxazolone	74-83	interleukin 18	Mus musculus	175-180	
27966619-9	2016	Compared to wild-type mice, NLRP3-/- mice exhibited higher sensitivity to oxazolone treatment with enhancement of Th2 cytokine expression and reduction of mature IL-1beta and IL-18 production; this phenotype was rescued by exogenous IL-1beta or IL-18.	Oxazolone	74-83	interleukin 18	Mus musculus	245-250	
11298831-8	2001	Importantly, not only was IL-18 able to contribute to the regulation of LC migration, it was found to be essential for the manifestation of these processes in response to topical sensitization with the contact allergen oxazolone.	Oxazolone	219-228	interleukin 18	Mus musculus	26-31