PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21118574-9	2010	RESULTS: We found that LPS-induced COX-2 expression and PGE2 production were mediated through NF-kappaB (p65) via activation of Toll-like receptor 4 (TLR4).	Dinoprostone	56-60	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	105-108	
27234497-12	2016	SaG2Fu and SaG4Fu also inhibited the activation and translocation of p65 NF-kappaB protein levels, resulting in reduction of NO, ROS, and PGE2 production.	Dinoprostone	138-142	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	69-82	
23317035-10	2013	PGE2, dbcAMP and 8-Cpt-cAMP induced the phosphorylation of Akt, IkappaB kinase and IkappaBalpha and the translocation of p65 to the nucleus and increased NF-kappaB dependent reporter gene activity, which was diminished by Akti.	Dinoprostone	0-4	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	121-124	
9862429-4	1998	PGE2 and iloprost also blocked disappearance of I kappaB-alpha from cytosolic fraction and nuclear translocation of NF-kappaB subunits p50 and p65.	Dinoprostone	0-4	v-rel reticuloendotheliosis viral oncogene homolog A (avian)	Mus musculus	143-146