PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20112284-6	2010	Moreover, COX-2-derived PGE(2) production appeared to involve in LPS-induced VCAM-1 expression which was attenuated by pretreatment with selective COX-2 inhibitors (NS-398 and celecoxib), transfection with COX-2 siRNA, or PGE(2) receptor antagonists.	Dinoprostone	24-30	vascular cell adhesion molecule 1	Homo sapiens	77-83	
7532667-5	1995	In addition, treatment with either isoproterenol or prostaglandin E2, which activates receptors coupled to Gs and increases [cAMP]i, also inhibited TNF-alpha-induced expression of ICAM-1 and VCAM-1 on ASM.	Dinoprostone	52-68	vascular cell adhesion molecule 1	Homo sapiens	191-197	
21867538-12	2011	Moreover, exogenous PGE2 also increased B16M cell adhesion to immobilized recombinant VCAM-1.	Dinoprostone	20-24	vascular cell adhesion molecule 1	Homo sapiens	86-92	
31200106-7	2019	Furthermore, treatment with PGE2 and PTZ increased IBA-1 (microglial marker), GFAP (astrocytic marker), 4-HNE (lipid peroxidation marker), VCAM-1 (vascular cell adhesion molecule 1), and p-PKAIIalpha (phosphorylated cAMP-dependent protein kinase) immunocontent.	Dinoprostone	28-32	vascular cell adhesion molecule 1	Homo sapiens	139-145	
31200106-7	2019	Furthermore, treatment with PGE2 and PTZ increased IBA-1 (microglial marker), GFAP (astrocytic marker), 4-HNE (lipid peroxidation marker), VCAM-1 (vascular cell adhesion molecule 1), and p-PKAIIalpha (phosphorylated cAMP-dependent protein kinase) immunocontent.	Dinoprostone	28-32	vascular cell adhesion molecule 1	Homo sapiens	147-180