PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28122338-3	2017	In this study, we found a cohort of miRNAs were dysregulated upon treatment with cisplatin, among of which miR-29b was the most upregulated one.	Cisplatin	81-90	microRNA 29b-1	Homo sapiens	107-114	
28122338-5	2017	However, upon treatment with cisplatin, the expression of miR-29b was significantly up-regulated.	Cisplatin	29-38	microRNA 29b-1	Homo sapiens	58-65	
28122338-7	2017	While, inhibition of miR-29b could prevent the cisplatin-induced epithelial features, cell movement and angiogenesis of CC cells, which means miR-29b/STAT3 axis participates in the chemotherapy of cisplatin in CC.	Cisplatin	47-56	microRNA 29b-1	Homo sapiens	21-28	
28122338-7	2017	While, inhibition of miR-29b could prevent the cisplatin-induced epithelial features, cell movement and angiogenesis of CC cells, which means miR-29b/STAT3 axis participates in the chemotherapy of cisplatin in CC.	Cisplatin	47-56	microRNA 29b-1	Homo sapiens	142-149	
28122338-7	2017	While, inhibition of miR-29b could prevent the cisplatin-induced epithelial features, cell movement and angiogenesis of CC cells, which means miR-29b/STAT3 axis participates in the chemotherapy of cisplatin in CC.	Cisplatin	197-206	microRNA 29b-1	Homo sapiens	21-28	
28122338-7	2017	While, inhibition of miR-29b could prevent the cisplatin-induced epithelial features, cell movement and angiogenesis of CC cells, which means miR-29b/STAT3 axis participates in the chemotherapy of cisplatin in CC.	Cisplatin	197-206	microRNA 29b-1	Homo sapiens	142-149	
26564501-0	2015	[miR-29b Reduces Cisplatin Resistance of Gastric Cancer Cell by Targeting PI3K/Akt Pathway].	Cisplatin	17-26	microRNA 29b-1	Homo sapiens	1-8	
26564501-1	2015	OBJECTIVE: To investigate the regulatory effect of miR-29b on gastric cells" resistance to cisplatin.	Cisplatin	91-100	microRNA 29b-1	Homo sapiens	51-58	
26564501-2	2015	METHODS: The expression of miR-29b in gastric cancer cell line treated with cisplatin concentration gradient was detected using quantitative reverse-transcriptase polymerase chain reaction (qRT-PCR) and Western blotting.	Cisplatin	76-85	microRNA 29b-1	Homo sapiens	27-34	
26564501-3	2015	CCK8 was used to measure the cell viability after cisplatin treatment in condition of miR-29b knock-down and overexpression.	Cisplatin	50-59	microRNA 29b-1	Homo sapiens	86-93	
26564501-4	2015	RESULTS: The expression of miR-29b was significantly upregualted by cisplatin treatment,while its target gene AKT2 was downregulated.	Cisplatin	68-77	microRNA 29b-1	Homo sapiens	27-34	
26564501-5	2015	The up-regulation of miR-29b enhanced the sensitivity of gastric cancer cells to cisplatin,while the knock-down of miR-29b enhanced the cisplatin resistance.	Cisplatin	81-90	microRNA 29b-1	Homo sapiens	21-28	
26564501-5	2015	The up-regulation of miR-29b enhanced the sensitivity of gastric cancer cells to cisplatin,while the knock-down of miR-29b enhanced the cisplatin resistance.	Cisplatin	136-145	microRNA 29b-1	Homo sapiens	115-122	
26564501-6	2015	Rescue experiments demonstrated that the miR-29b might regulate cisplatin resistance of gastric cancer cell by targeting PI3K/Akt pathway.	Cisplatin	64-73	microRNA 29b-1	Homo sapiens	41-48	
25784815-12	2015	Upregulation of miR-29b also enhanced the chemosensitivity of PCa cells to cisplatin.	Cisplatin	75-84	microRNA 29b-1	Homo sapiens	16-23	
27199349-8	2016	We also noted that miR-29b could confer sensitivity to intrinsic apoptosis triggered by exposure to cisplatin, a drug used widely in lung cancer treatment.	Cisplatin	100-109	microRNA 29b-1	Homo sapiens	19-26	
23591808-6	2013	In addition, the IC50 for cisplatin in the miR-29b-treated cells was effectively reduced.	Cisplatin	26-35	microRNA 29b-1	Homo sapiens	43-50