PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29311617-2	2018	PYY production is highest in enteroendocrine cells located in the distal intestine, mirroring the sites where high concentrations of short chain fatty acids (SCFAs) are produced by gut microbiota.	Fatty Acids, Volatile	133-156	peptide YY	Homo sapiens	0-3	
32383415-4	2020	This process yields SCFA which stimulate the release of appetite-suppressing hormones glucagon-like peptide-1 and peptide YY.	Fatty Acids, Volatile	20-24	peptide YY	Homo sapiens	114-124	
24997035-9	2014	The function of PYY-releasing enteroendocrine cells is directly influenced by short chain fatty acids generated by the intestinal microbiota from indigestible fibre, while NPY may control the impact of the gut microbiota on inflammatory processes, pain, brain function and behaviour.	Fatty Acids, Volatile	78-101	peptide YY	Homo sapiens	16-19	
28539646-7	2017	Colonic infusions of SCFA mixtures, in concentrations and ratios reached after fibre intake, increased fat oxidation, energy expenditure and PYY, and decreased lipolysis in overweight/obese men.	Fatty Acids, Volatile	21-25	peptide YY	Homo sapiens	141-144	
8345132-5	1993	As peptide YY slows colonic transit, it could also improve salvage of unabsorbed nutrients by enhancing bacterial production of volatile fatty acids and their subsequent absorption by the colonic mucosa.	Fatty Acids, Volatile	128-148	peptide YY	Homo sapiens	3-13	
34603471-5	2021	Gut microbiota can modulate gut functions through the metabolites of bacterial fermentation, among which short-chain fatty acids (SCFAs), secondary bile salts (BAs), and methane occupied more important positions and could trigger the release of gut hormones from enteroendocrine cells (EECs), such as 5-hydroxytryptamine (5-HT), peptide YY (PYY), and glucagon-like peptide-1 (GLP-1).	Fatty Acids, Volatile	105-128	peptide YY	Homo sapiens	329-339	
34603471-5	2021	Gut microbiota can modulate gut functions through the metabolites of bacterial fermentation, among which short-chain fatty acids (SCFAs), secondary bile salts (BAs), and methane occupied more important positions and could trigger the release of gut hormones from enteroendocrine cells (EECs), such as 5-hydroxytryptamine (5-HT), peptide YY (PYY), and glucagon-like peptide-1 (GLP-1).	Fatty Acids, Volatile	105-128	peptide YY	Homo sapiens	341-344	
18931303-6	2008	Functional genomic, biochemical, and physiologic studies of germ-free and cocolonized Gpr41-/- and +/+ littermates disclosed that Gpr41-deficiency is associated with reduced expression of PYY, an enteroendocrine cell-derived hormone that normally inhibits gut motility, increased intestinal transit rate, and reduced harvest of energy (short-chain fatty acids) from the diet.	Fatty Acids, Volatile	336-359	peptide YY	Homo sapiens	188-191	
16687037-4	2006	PYY release is stimulated by intraluminal nutrients, including glucose, bile salts, lipids, short-chain fatty acids and amino acids.	Fatty Acids, Volatile	92-115	peptide YY	Homo sapiens	0-3