PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9631239-9	1998	In summary, RV-specific downregulation of PDE-3A mRNA/protein(s) at 3 wk suggests that hemodynamic rather than humoral mechanisms are responsible, and provides a molecular basis for the limited efficacy of milrinone in the progression of HF.	Milrinone	206-215	phosphodiesterase 3A	Canis lupus familiaris	42-48	
9386183-0	1997	Development of decompensated dilated cardiomyopathy is associated with decreased gene expression and activity of the milrinone-sensitive cAMP phosphodiesterase PDE3A.	Milrinone	117-126	phosphodiesterase 3A	Canis lupus familiaris	160-165	
9386183-11	1997	CONCLUSIONS: Selective downregulation of PDE3A may account in part for the ineffectiveness of milrinone in the treatment of severe CHF.	Milrinone	94-103	phosphodiesterase 3A	Canis lupus familiaris	41-46