PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2502045-5	1989	Unlike Hu-IFN-alpha A, but similar to Hu-IFN-gamma, the hybrid Hu-IFN-alpha A/gamma can be phosphorylated by [gamma 32P]ATP and cAMP-dependent protein kinase.	Adenosine Triphosphate	120-123	interferon alpha 1	Homo sapiens	66-75	
34256772-10	2021	Interestingly, IFN-alpha is capable of inducing ROS and ATP production in CD4+ T cells, while knockdown of tRF-3009 reversed this process.	Adenosine Triphosphate	56-59	interferon alpha 1	Homo sapiens	15-24	
1691881-3	1990	The main effect of this IFN induced enzyme activation is the production of oligonucleotides (2-5A) from cellular ATP.	Adenosine Triphosphate	113-116	interferon alpha 1	Homo sapiens	24-27	
25387665-5	2015	RESULTS: JTE-052 inhibited the JAK1, JAK2, JAK3, and tyrosine kinase (Tyk)2 enzymes in an adenosine triphosphate (ATP)-competitive manner and inhibited cytokine signaling evoked by IL-2, IL-6, IL-23, granulocyte/macrophage colony-stimulating factor, and IFN-alpha.	Adenosine Triphosphate	114-117	interferon alpha 1	Homo sapiens	254-263	
25891073-4	2015	Furthermore, truncated RIG-I/MDA5 lacking the signaling domain, and hence the IFN stimulatory activity, displaces viral proteins and suppresses replication of certain viruses in an ATP-dependent manner.	Adenosine Triphosphate	181-184	interferon alpha 1	Homo sapiens	78-81	
6435868-1	1984	2",5"-Oligoadenylate (2-5A) synthetase, which polymerizes adenosine triphosphate into 2-5A, is induced upon treatment of cells with interferon (IFN) and is thought to be involved in its antiviral and anticellular action.	Adenosine Triphosphate	58-80	interferon alpha 1	Homo sapiens	132-148	
17535916-2	2007	dsRNA produced during viral infections activates IFN-inducible synthetases that produce 5"-phosphorylated, 2",5"-oligoadenylates (2-5A) from ATP.	Adenosine Triphosphate	141-144	interferon alpha 1	Homo sapiens	49-52	
15548813-5	2004	The ATP chemosensitivity assay was used to study sensitivity to IFN-alpha-2b in freshly isolated cells from each tumour.	Adenosine Triphosphate	4-7	interferon alpha 1	Homo sapiens	64-73	
15121865-7	2004	Further analyses indicated that the BAF complex primed some IFN target promoters by utilizing ATP-derived energy to maintain the chromatin in a constitutively open conformation, allowing faster and more potent induction after IFN-alpha treatment.	Adenosine Triphosphate	94-97	interferon alpha 1	Homo sapiens	60-63	
15121865-7	2004	Further analyses indicated that the BAF complex primed some IFN target promoters by utilizing ATP-derived energy to maintain the chromatin in a constitutively open conformation, allowing faster and more potent induction after IFN-alpha treatment.	Adenosine Triphosphate	94-97	interferon alpha 1	Homo sapiens	226-235	
22299633-6	2012	When human EPCs were stimulated with LPS and nucleotides, ATP or UTP inhibited the expression of pro-inflammatory cytokines including MCP-1 (monocyte chemoattractant protein-1), IFNalpha (interferon alpha), TNFalpha (tumour necrosis factor alpha) and adhesion molecule VCAM-1 (vascular cell adhesion molecule 1) induced by LPS.	Adenosine Triphosphate	58-61	interferon alpha 1	Homo sapiens	178-186	
22264514-4	2012	We show that IFNalpha protects human lung epithelial cells from alpha-toxin-induced intracellular ATP depletion and cell death by reducing extracellular ATP leakage.	Adenosine Triphosphate	98-101	interferon alpha 1	Homo sapiens	13-21	
22264514-4	2012	We show that IFNalpha protects human lung epithelial cells from alpha-toxin-induced intracellular ATP depletion and cell death by reducing extracellular ATP leakage.	Adenosine Triphosphate	153-156	interferon alpha 1	Homo sapiens	13-21