PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28404626-7	2017	Expression analysis by quantitative reverse transcription polymerase chain reaction of extracellular purine degrading enzymes and P1 and P2 receptors on T cells isolated from the injured heart revealed profound upregulation of the enzymatic machinery for hydrolysis of extracellular adenosine triphosphate and nicotinamide adenine dinucleotide, both pathways converging in the formation of AMP and adenosine via CD73.	Adenosine Triphosphate	283-305	zinc finger protein 185	Mus musculus	130-139	
11560866-4	2001	Incubation of SVA with mouse liver preparations fortified with CoASH and ATP led to formation of SV and two major products (P1 and P2).	Adenosine Triphosphate	73-76	zinc finger protein 185	Mus musculus	124-133	
10694235-2	2000	We have previously shown that ATP increased cyclic AMP in NG108-15 cells, which was inhibited by P(1) receptor antagonist methylxanthines.	Adenosine Triphosphate	30-33	zinc finger protein 185	Mus musculus	97-101	
10694235-19	2000	These results clearly demonstrate that ATP-stimulated cyclic AMP formation can be distinguished from the A(2A) receptor agonist-induced one by using the several P(1) and P2 receptor antagonists.	Adenosine Triphosphate	39-42	zinc finger protein 185	Mus musculus	161-165	
19549027-9	2009	In an ex vivo model of the murine trachea we were able to show that Der p 1, in contrast to Phl p 1, enhances the transportation velocity of particles by the trachea, presumably by ATP released from the injured epithelium.	Adenosine Triphosphate	181-184	zinc finger protein 185	Mus musculus	72-75