PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28432138-4	2017	Inhibition of Drp1 alleviates mitochondrial fragmentation, loss of mitochondrial membrane potential, reactive oxygen species production, ATP reduction, and synaptic depression in Abeta-treated neurons.	Adenosine Triphosphate	137-140	dynamin 1-like	Mus musculus	14-18	
32521505-9	2020	In particular, the Dnm1l knockout most significantly affected mitochondrial morphology, energy metabolism, and ATP production in ESCs.	Adenosine Triphosphate	111-114	dynamin 1-like	Mus musculus	19-24	
29673648-6	2018	However, inhibition of Drp1 by Mdivi1 (Drp1 inhibitor) resulted in higher OxPhos ATP production and lower mitochondrial membrane potential.	Adenosine Triphosphate	81-84	dynamin 1-like	Mus musculus	23-27	
29673648-6	2018	However, inhibition of Drp1 by Mdivi1 (Drp1 inhibitor) resulted in higher OxPhos ATP production and lower mitochondrial membrane potential.	Adenosine Triphosphate	81-84	dynamin 1-like	Mus musculus	39-43	
29166700-5	2018	Suppression of DLP1 expression by DPL1 RNAi inhibited prion-induced mitochondrial fragmentation and dysfunction (measured by ADP/ATP ratio, mitochondrial membrane potential, and mitochondrial integrity).	Adenosine Triphosphate	129-132	dynamin 1-like	Mus musculus	15-19	
25880092-8	2015	Although Drp1KO increased the distance between axonal mitochondria, mitochondrial-derived ATP still decreased similarly in Drp1KO boutons with and without mitochondria.	Adenosine Triphosphate	90-93	dynamin 1-like	Mus musculus	123-129	
26825530-6	2016	When isolated from diabetic mice and cultured in high glucose, Drp1-null podocytes had more elongated mitochondria and better mitochondrial fitness associated with enhanced oxygen consumption and ATP production than wild-type podocytes.	Adenosine Triphosphate	196-199	dynamin 1-like	Mus musculus	63-67	
26370078-9	2015	In conclusion, failure of Drp1 disassembly impairs mitophagy, leading to a downstream cascade of mitochondrial depolarization, aberrant calcium handling, impaired ATP synthesis, and activation of sterile myocardial inflammation, resulting in heart failure.	Adenosine Triphosphate	163-166	dynamin 1-like	Mus musculus	26-30	
25880092-9	2015	This indicates that mitochondrial-derived ATP is rapidly dispersed in Drp1KO axons, and that the deficits in axonal bioenergetics and function are not caused by regional energy gradients.	Adenosine Triphosphate	42-45	dynamin 1-like	Mus musculus	70-76	
34310799-6	2021	In contrast, the transfection of ALR into LEPCs significantly inhibited Drp1 phosphorylation, thereby favoring the maintenance of mitochondrial integrity and the preservation of ATP contents in LEPCs.	Adenosine Triphosphate	178-181	dynamin 1-like	Mus musculus	72-76	
24284040-6	2014	Drp-1 siRNA transfection preserved the mitochondrial membrane potential (MMP), reduced cytochrome c release, enhanced ATP production, and partly prevented mitochondrial swelling.	Adenosine Triphosphate	118-121	dynamin 1-like	Mus musculus	0-5	
19752021-4	2009	This embryonic lethality is not likely caused by gross energy deprivation, as Drp1-null cells showed normal intracellular adenosine triphosphate levels.	Adenosine Triphosphate	122-144	dynamin 1-like	Mus musculus	78-82