PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 7004861-6 1981 From the above results it may be concluded that angiotensinogen production is dependent on thyroid hormones and that renin release depends on beta-adrenergic receptor sensitivity to catecholamines, which is reduced by thyroidectomy. Catecholamines 182-196 renin Rattus norvegicus 117-122 3285225-6 1988 An increase in circulating plasma catecholamine concentrations appeared to mediate yohimbine-induced renin release since propranolol prevented the rise in PRA caused by yohimbine in renal denervated rats. Catecholamines 34-47 renin Rattus norvegicus 101-106 6149822-9 1984 These data suggest that digitalis agents can interact with sites in the central nervous system to induce a release of renin from the kidney; this release appears to involve activation of beta-adrenergic receptors by catecholamines from the adrenal medulla, perhaps through a direct adrenal-kidney vascular network. Catecholamines 216-230 renin Rattus norvegicus 118-123 6186863-1 1983 The potential role of circulating catecholamines in the regulation of plasma renin concentration (PRC) was evaluated in conscious rats. Catecholamines 34-48 renin Rattus norvegicus 77-82 6186863-5 1983 It is concluded that, in the rat, the circulating catecholamines contribute to the control of PRC by modulating renin secretion through stimulation-inhibition mechanisms. Catecholamines 50-64 renin Rattus norvegicus 112-117 6292084-9 1982 The renin-stimulating effect of centrally administered PGE2 is, at least in part, dependent on beta-adrenergic receptor stimulation by increased circulating catecholamines. Catecholamines 157-171 renin Rattus norvegicus 4-9 6175808-1 1982 Acute hypovolemia induced by bleeding (5 ml/300 g body weight) halothane-anesthetized (0.8% in oxygen) rats is attended by hypotension, bradycardia, and increases in plasma renin, vasopressin, and catecholamine levels. Catecholamines 197-210 renin Rattus norvegicus 173-178 6658140-7 1983 The relationship between the renin-AII system and the central nervous system catecholamines could be involved in the control of development and maintenance of the renal arterial hypertension. Catecholamines 77-91 renin Rattus norvegicus 29-34 7418541-3 1980 The main purpose of the study was to find out how a reduction or an increase of total body sodium and the associated changes of renin production can influence the vascular response to angiotensin and to catecholamines. Catecholamines 203-217 renin Rattus norvegicus 128-133 7004910-1 1980 It has been established that the catecholamine-induced increase in renin secretion by juxtaglomerular apparatus cells and sodium reabsorption stimulation in the rat kidney are consequent on the excitation of renal beta-adrenoreceptors. Catecholamines 33-46 renin Rattus norvegicus 67-72 7004910-3 1980 When given in a dose inhibiting angiotensin II formation and renin-secreting effect of catecholamines, heparin also diminishes their activating effect on tubular sodium transport. Catecholamines 87-101 renin Rattus norvegicus 61-66 7004910-4 1980 It is suggested that the renin-angiotensin system may be directly involved into the mechanism of catecholamine-stimulated sodium reabsorption by the rat kidney. Catecholamines 97-110 renin Rattus norvegicus 25-30 673021-0 1978 Inhibitory effect of tyramine-induced release of catecholamines on renin secretion. Catecholamines 49-63 renin Rattus norvegicus 67-72 673021-6 1978 It is concluded that catecholamines which are released from renal sympathetic nerve endings can suppress renin release by activating alpha-adrenoceptors. Catecholamines 21-35 renin Rattus norvegicus 105-110 1015916-0 1976 Effect of some antihypertensive drugs and catecholamine depletors on the plasma renin activity in the rat. Catecholamines 42-55 renin Rattus norvegicus 80-85 215928-9 1978 These data indicate that sodium deprivation enhances the sensitivity of the renin-secreting cells to catecholamine stimulation, and are consistent with the hypothesis that the increase in renin secretion produced by NE is mediated via cyclic AMP. Catecholamines 101-114 renin Rattus norvegicus 76-81 199532-4 1977 In these investigations it has been tested to see whether the catecholamines released by this activation modulate renin release by stimulation of certain alpha-receptors. Catecholamines 62-76 renin Rattus norvegicus 114-119 199532-8 1977 It is concluded that catecholamines released from the sympathetic nervous system can decrease renin secretion by an activation of certain alpha-receptors. Catecholamines 21-35 renin Rattus norvegicus 94-99 1071719-12 1976 Catecholamines stimulated renin release in vitro in proportion to the potency of their action on beta-adrenoreceptors. Catecholamines 0-14 renin Rattus norvegicus 26-31 1015916-1 1976 The effect of some antihypertensive drugs and catecholamine depletors on the plasma renin activity (PRA) has been investigated in the rat. Catecholamines 46-59 renin Rattus norvegicus 84-89 1204291-10 1975 These observations suggest that catecholamines stimulate renin secretion by an intrarenal effect which is largely independent of changes in renal perfusion pressure. Catecholamines 32-46 renin Rattus norvegicus 57-62 4370333-0 1974 Renin release by rat kidney slices in vitro: effects of cations and catecholamines. Catecholamines 68-82 renin Rattus norvegicus 0-5 1236608-8 1975 Since the vasoconstrictor effect of clonidine is considered to be due to alpha-adrenergic stimulation, this observation is similar to a previous study reporting suppression of renin secretion by vasoconstrictor infusions of catecholamines (Vandongen & Peart, 1974). Catecholamines 224-238 renin Rattus norvegicus 176-181 1157220-1 1975 Controversy exists regarding the mechanism by which catecholamines stimulate renin secretion in vivo. Catecholamines 52-66 renin Rattus norvegicus 77-82 21785727-5 2011 Moreover, increased activity of the renin-angiotensin and sympathetic nervous systems leading to downregulation of receptors may be responsible for the blunted vascular sensitivity to angiotensin II and catecholamines, respectively. Catecholamines 203-217 renin Rattus norvegicus 36-41 9736275-5 1998 Because norepinephrine increased 1.7-fold and 3.2-fold and plasma epinephrine increased 3.9-fold and 7.8-fold during hypoxia and CO inhalation, respectively, circulating catecholamines might mediate the stimulatory effects of hypoxia on renin secretion and renin gene expression. Catecholamines 170-184 renin Rattus norvegicus 237-242 9736275-5 1998 Because norepinephrine increased 1.7-fold and 3.2-fold and plasma epinephrine increased 3.9-fold and 7.8-fold during hypoxia and CO inhalation, respectively, circulating catecholamines might mediate the stimulatory effects of hypoxia on renin secretion and renin gene expression. Catecholamines 170-184 renin Rattus norvegicus 257-262 9140020-1 1997 This study was designed to determine if the increase in plasma renin activity (PRA) that occurs during water deprivation is mediated by the renal sympathetic nerves or adrenomedullary catecholamine release. Catecholamines 184-197 renin Rattus norvegicus 63-68 8156394-0 1994 Brain catecholamines mediate the delayed reduction in renin release after injection of fenfluramine. Catecholamines 6-20 renin Rattus norvegicus 54-59 1823883-13 1991 It is most likely that the adrenal renin plays a role in the production of aldosterone in the adrenal cortex or in the secretion of catecholamines from the adrenal medulla through intra- and/or extracellular formation of angiotensin II. Catecholamines 132-146 renin Rattus norvegicus 35-40 12621527-9 2003 These findings provide evidence that catecholamines are involved in mechanisms that rapidly alter the expression of the genes of the renin-angiotensin system within the kidney. Catecholamines 37-51 renin Rattus norvegicus 133-138