PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25161195-0	2014	Adipocyte ALK7 links nutrient overload to catecholamine resistance in obesity.	Catecholamines	42-55	activin A receptor, type IC	Mus musculus	10-14	
25161195-2	2014	We report that targeted disruption of TGF-beta superfamily receptor ALK7 alleviates diet-induced catecholamine resistance in adipose tissue, thereby reducing obesity in mice.	Catecholamines	97-110	activin A receptor, type IC	Mus musculus	68-72	
34245608-3	2021	Constitutive ablation of the activin receptor ALK7 in adipose tissue enhances catecholamine signaling and lipolysis in adipocytes, and protects mice from diet-induced obesity.	Catecholamines	78-91	activin A receptor, type IC	Mus musculus	46-50	
34245608-7	2021	Mechanistically, combination of ALK7 ablation with either treatment strongly enhanced the levels of beta3-AR, the main adrenergic receptor for catecholamine stimulation of lipolysis, and C/EBPalpha, an upstream regulator of beta3-AR expression.	Catecholamines	143-156	activin A receptor, type IC	Mus musculus	32-36