PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15944259-5	2005	In the present study, we showed that TCR engagement does not influence hypoxia-dependent stabilization but stimulates protein synthesis of HIF-1alpha, most possibly via PI3K/mammalian target of rapamycin system, and that expression of HIF-1alpha and its target genes is blocked by treatment with rapamycin.	Sirolimus	194-203	T cell receptor beta variable 20/OR9-2 (non-functional)	Homo sapiens	37-40	
15067056-9	2004	Based on these data, we propose that the decision as to whether TCR engagement will lead to productive activation or tolerance is dictated by a rapamycin -inhibitable pathway, independent of the G(1)-->S phase cell cycle progression.	Sirolimus	144-153	T cell receptor beta variable 20/OR9-2 (non-functional)	Homo sapiens	64-67	
15162441-5	2004	While blockade of TCR signaling pathways with inhibitors of the phosphatidylinositol 3-kinase pathway caused a partial pre-integration block, another inhibitor, rapamycin, completely suppressed the infection.	Sirolimus	161-170	T cell receptor beta variable 20/OR9-2 (non-functional)	Homo sapiens	18-21	
8660826-5	1996	Prevention of cell death correlated with delayed entry into S phase from G1 following TCR religation in the rapamycin-treated cultures.	Sirolimus	108-117	T cell receptor beta variable 20/OR9-2 (non-functional)	Homo sapiens	86-89