PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 32397282-4 2020 Rapamycin treatment was started either at day 1 or at day 7 of BAPN+AngII challenge, and continued throughout the observational period. Sirolimus 0-9 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 68-73 30074157-0 2018 Rapamycin Treatment Attenuates Angiotensin II -induced Abdominal Aortic Aneurysm Formation via VSMC Phenotypic Modulation and Down-regulation of ERK1/2 Activity. Sirolimus 0-9 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 31-45 30074157-11 2018 Moreover, rapamycin reversed Ang II -induced VSMCs phenotypic change both in vivo and in vitro. Sirolimus 10-19 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 29-35 27254006-6 2016 To test the functional role of brain mTORC1 in mediating the action of Ang II, we examined the consequence of mTORC1 inhibition with rapamycin on Ang II-induced increase in water intake and arterial pressure. Sirolimus 133-142 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 146-152 27254006-7 2016 ICV pretreatment with rapamycin blocked ICV Ang II-mediated increases in the frequency, duration, and amount of water intake but did not interfere with the pressor response evoked by Ang II. Sirolimus 22-31 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 44-50 26562437-8 2016 Furthermore, in C57 BL/6 mice with Ang II infusion, intraperitoneal administration of Rapa ameliorated Ang II-induced cardiac fibrosis and cardiac dysfunction, while CQ treatment not only exacerbated Ang II-mediated cardiac fibrosis and cardiac dysfunction, but also impaired cardiac function. Sirolimus 86-90 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 35-41 26562437-8 2016 Furthermore, in C57 BL/6 mice with Ang II infusion, intraperitoneal administration of Rapa ameliorated Ang II-induced cardiac fibrosis and cardiac dysfunction, while CQ treatment not only exacerbated Ang II-mediated cardiac fibrosis and cardiac dysfunction, but also impaired cardiac function. Sirolimus 86-90 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 103-109 26562437-8 2016 Furthermore, in C57 BL/6 mice with Ang II infusion, intraperitoneal administration of Rapa ameliorated Ang II-induced cardiac fibrosis and cardiac dysfunction, while CQ treatment not only exacerbated Ang II-mediated cardiac fibrosis and cardiac dysfunction, but also impaired cardiac function. Sirolimus 86-90 angiotensinogen (serpin peptidase inhibitor, clade A, member 8) Mus musculus 103-109