PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30629952-0	2019	The aggravating effect of selenium deficiency on T-2 toxin-induced damage on primary cardiomyocyte results from a reduction of protective autophagy.	Selenium	26-34	solute carrier family 25 member 5	Homo sapiens	49-52	
30629952-2	2019	The aim of this study was to explore the role of autophagy in the aggravation of selenium deficiency on T-2 toxin-induced damages on primary cardiomyocyte.	Selenium	81-89	solute carrier family 25 member 5	Homo sapiens	104-107	
30629952-3	2019	Our present study demonstrated that 0.25-1 muM T-2 toxin damaged primary cardiomyocytes and selenium deficiency exacerbated T-2 toxin-induced damages by measuring the levels of MTT, lactate dehydrogenase and cleaved-caspase 3.	Selenium	92-100	solute carrier family 25 member 5	Homo sapiens	124-127	
30629952-6	2019	Selenium deficiency lowered cytoprotective autophagy in the primary cardiomyocytes treated by T-2 toxin.	Selenium	0-8	solute carrier family 25 member 5	Homo sapiens	94-97	
30629952-7	2019	It can be concluded that autophagy induced by T-2 toxin plays a role in protecting primary cardiomyocyte, but selenium deficiency decreases the protective autophagy and then exacerbate T-2 toxin-induced damages.	Selenium	110-118	solute carrier family 25 member 5	Homo sapiens	185-188	
28965579-0	2017	Selenium promotes metabolic conversion of T-2 toxin to HT-2 toxin in cultured human chondrocytes.	Selenium	0-8	solute carrier family 25 member 5	Homo sapiens	42-45	
28965579-10	2017	Supplementary selenium in the HCs cultures reduced the cytotoxicity induced by T-2 toxin significantly, and was associated with rapid conversion of T-2 toxin in the culture medium to HT-2 toxin.	Selenium	14-22	solute carrier family 25 member 5	Homo sapiens	79-82	
28965579-10	2017	Supplementary selenium in the HCs cultures reduced the cytotoxicity induced by T-2 toxin significantly, and was associated with rapid conversion of T-2 toxin in the culture medium to HT-2 toxin.	Selenium	14-22	solute carrier family 25 member 5	Homo sapiens	148-151	
25264878-7	2014	Interestingly, Selenium (Se) can partly block T-2 toxin -induced mitochondria dysfunction, oxidative damage and chondrocytes apoptosis.	Selenium	15-23	solute carrier family 25 member 5	Homo sapiens	46-49	
25264878-7	2014	Interestingly, Selenium (Se) can partly block T-2 toxin -induced mitochondria dysfunction, oxidative damage and chondrocytes apoptosis.	Selenium	15-17	solute carrier family 25 member 5	Homo sapiens	46-49	
21144892-4	2011	Immunohistochemistry analyses showed that T-2 toxin decreased type II collagen staining and selenium appeared to prevent the decrease in type II collagen induced by T-2 toxin in engineered cartilage.	Selenium	92-100	solute carrier family 25 member 5	Homo sapiens	165-168	
18196609-12	2008	Selenium could partly inhibit the effects of T-2 toxin above.	Selenium	0-8	solute carrier family 25 member 5	Homo sapiens	45-48	
16307838-0	2006	T-2 toxin induces apoptosis, and selenium partly blocks, T-2 toxin induced apoptosis in chondrocytes through modulation of the Bax/Bcl-2 ratio.	Selenium	33-41	solute carrier family 25 member 5	Homo sapiens	57-60	
16307838-8	2006	Selenium could partly block the apoptosis of chondrocytes induced by T-2 toxin through decreasing the Bax/Bcl-2 ratio.	Selenium	0-8	solute carrier family 25 member 5	Homo sapiens	69-72	
16624732-0	2006	[Protective effect of selenium against T-2 toxin-induced inhibition of chondrocyte aggrecan and collagen II synthesis].	Selenium	22-30	solute carrier family 25 member 5	Homo sapiens	39-42	
16624732-1	2006	OBJECTIVE: To study the inhibitory effect of T-2 toxin on the expression of aggrecan and collagen II in chondrocytes and the protection of selenium against this effect.	Selenium	139-147	solute carrier family 25 member 5	Homo sapiens	45-48	
16624732-7	2006	Selenium could partly attenuate the inhibitory effects of T-2 toxin on aggrecan mRNA expression, but showed no such effect against T-2-induced collagen II expression.	Selenium	0-8	solute carrier family 25 member 5	Homo sapiens	58-61	
16624732-8	2006	CONCLUSION: T-2 toxin can obviously inhibit aggrecan and collagen II synthesis in human chondrocytes, and selenium can partly antagonize the inhibitory effects of T-2 toxin on aggrecan.	Selenium	106-114	solute carrier family 25 member 5	Homo sapiens	163-166