PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
3768091-4	1986	At a further increase of total HDL-chol, cholesterol is increasingly isolated in the HDL2-fraction, especially when HDL3-chol has reached its maximum (about 1.25 mmol/l).	chol	35-39	junctophilin 3	Homo sapiens	85-89	
3300667-7	1987	The rise in HDL-chol was accounted for by an increase in both HDL2-chol and HDL3-chol subfractions.	chol	16-20	junctophilin 3	Homo sapiens	62-66	
3300667-7	1987	The rise in HDL-chol was accounted for by an increase in both HDL2-chol and HDL3-chol subfractions.	chol	67-71	junctophilin 3	Homo sapiens	62-66	
3300667-7	1987	The rise in HDL-chol was accounted for by an increase in both HDL2-chol and HDL3-chol subfractions.	chol	67-71	junctophilin 3	Homo sapiens	62-66	
3300667-8	1987	Following the shift from CIT to CSII, HDL2-chol rose further (P less than 0.05), whereas HDL3-chol remained unchanged.	chol	43-47	junctophilin 3	Homo sapiens	38-42	
3300667-9	1987	When CSII was changed to CIT, the HDL3-chol level decreased (P less than 0.02), but HDL2-chol remained constant.	chol	89-93	junctophilin 3	Homo sapiens	84-88	
3300667-11	1987	HDL2-chol was 35% higher in females when compared to males at entry, and it rose in both sexes during CSII.	chol	5-9	junctophilin 3	Homo sapiens	0-4	
3300667-14	1987	Thus, even a mild improvement in glycemic control during CSII is associated with a rise in HDL-chol, particularly the HDL2 subfraction.	chol	95-99	junctophilin 3	Homo sapiens	118-122	
2507376-3	1989	Although HDL2-chol was similar in diabetic and control groups, the HDL2-chol-to-free-cholesterol ratio (diabetic vs. control, 4.64 +/- 1.7 vs. 1.96 +/- 1.0 mumol/ml, P less than .025) and the sphingomyelin-to-lecithin ratio (0.23 +/- 0.08 vs. 0.20 +/- 0.09, P less than .025) were both significantly increased in the IDDM group.	chol	14-18	junctophilin 3	Homo sapiens	9-13	
2507376-3	1989	Although HDL2-chol was similar in diabetic and control groups, the HDL2-chol-to-free-cholesterol ratio (diabetic vs. control, 4.64 +/- 1.7 vs. 1.96 +/- 1.0 mumol/ml, P less than .025) and the sphingomyelin-to-lecithin ratio (0.23 +/- 0.08 vs. 0.20 +/- 0.09, P less than .025) were both significantly increased in the IDDM group.	chol	72-76	junctophilin 3	Homo sapiens	67-71	
3768091-4	1986	At a further increase of total HDL-chol, cholesterol is increasingly isolated in the HDL2-fraction, especially when HDL3-chol has reached its maximum (about 1.25 mmol/l).	chol	41-45	junctophilin 3	Homo sapiens	85-89	
3768091-5	1986	Thus, the magnitude of the absolute intra-individual variation of either HDL3-chol or HDL2-chol (in mmol/l) is related to the total HDL-chol concentration.	chol	91-95	junctophilin 3	Homo sapiens	86-90	
3768091-5	1986	Thus, the magnitude of the absolute intra-individual variation of either HDL3-chol or HDL2-chol (in mmol/l) is related to the total HDL-chol concentration.	chol	91-95	junctophilin 3	Homo sapiens	86-90	
6738685-6	1984	The HDL2-chol decreased in women.	chol	9-13	junctophilin 3	Homo sapiens	4-8	
22584154-1	2012	INTRODUCTION: Higher levels of high density lipoprotein (HDL) subfractions HDL3-chol and particularly HDL2-chol protect against cardiovascular disease (CVD), but inflammation reduces the HDL level and may impair its anti-atherogenic effect.	chol	107-111	junctophilin 3	Homo sapiens	102-106	
22584154-13	2012	CONCLUSIONS: Both HDL subfractions but particularly HDL2-chol concentrations were decreased in RA, primarily in women.	chol	57-61	junctophilin 3	Homo sapiens	52-56	
22584154-15	2012	The reduction of the HDL subfraction concentrations, particularly the supposedly beneficial HDL2-chol, may negatively impact the cardiovascular risk profile of women with RA.	chol	97-101	junctophilin 3	Homo sapiens	92-96	
22584154-3	2012	In this study, we investigated whether HDL2-chol and HDL3-chol concentrations differ between RA patients and healthy controls, and whether these levels are related to the level of RA disease activity.	chol	44-48	junctophilin 3	Homo sapiens	39-43	
22584154-6	2012	HDL2-chol and HDL3-chol concentrations were obtained by ultracentrifugation.	chol	5-9	junctophilin 3	Homo sapiens	0-4	
22584154-7	2012	Regression modeling was used to compare HDL subfraction levels between RA patients and healthy controls, and to analyze the effect of disease activity on HDL2-chol and HDL3-chol.	chol	159-163	junctophilin 3	Homo sapiens	154-158	
22584154-8	2012	RESULTS: HDL2-chol and HDL3-chol were significantly lower in RA patients compared to healthy controls (P = 0.01, P = 0.005, respectively).	chol	14-18	junctophilin 3	Homo sapiens	9-13	
22584154-10	2012	Reduced HDL2-chol and HDL3-chol levels were primarily present in female RA patients and not in male RA patients.	chol	13-17	junctophilin 3	Homo sapiens	8-12	
22584154-11	2012	A modest effect of the disease activity score in 28 joins ( DAS28) on HDL2-chol concentrations was found, after correction for disease duration, glucocorticosteroid use and body mass index (BMI), with a 0.06 mmol/L decrease with every point increase in DAS28 (P = 0.05).	chol	75-79	junctophilin 3	Homo sapiens	70-74