PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 31512297-10 2019 The difference (Delta) between the tau V O 2 p and MRT-TOI was greater during U VH compared to U M (12 +- 7 vs. 2 +- 7 s, P < 0.001) and during M VH (23 +- 15 s) compared to other conditions (P < 0.02), suggesting an increased proportional speeding of fractional O2 extraction. Oxygen 274-276 microtubule associated protein tau Homo sapiens 35-38 33536736-6 2020 O2 uptake (Vo2 ) and CO2 output (Vco2 ) off-kinetics was examined using a mono-exponential model in which tau off (tauoff ) and mean response time (MRT) were determined. Oxygen 0-2 microtubule associated protein tau Homo sapiens 106-109 34738228-9 2021 A higher amount of inactivity was associated with a lower peak oxygen uptake (correlation coefficient tau -0.48, p=0.023). Oxygen 63-69 microtubule associated protein tau Homo sapiens 102-105 24439479-5 2014 Additionally, among ApoE3+ subjects, the apnea and/or hypopnea with 4% O2-desaturation index was positively correlated with p-tau (r = 0.30, p = 0.023), t-tau (r = 0.31, p = 0.021), and Abeta-42 (r = 0.31, p = 0.021). Oxygen 71-73 microtubule associated protein tau Homo sapiens 126-129 8052661-5 1994 AGE-recombinant tau generated reactive oxygen intermediates and, when introduced into the cytoplasm of SH-SY5Y neuroblastoma cells, induced oxidant stress. Oxygen 39-45 microtubule associated protein tau Homo sapiens 16-19 19542619-4 2009 Examination of an in vitro model of cultured rat corticohippocampal neurons revealed that even relatively low level expression of human truncated tau protein (equal to 50% of endogenous tau) induced oxidative stress that resulted in increased depolarization of mitochondria (approximately 1.2-fold above control, P < 0.01) and increases in reactive oxygen species (approximately 1.3-fold above control, P < 0.001). Oxygen 349-355 microtubule associated protein tau Homo sapiens 146-149 19542619-6 2009 Furthermore, using two common antioxidants (vitamin C and E), we were able significantly eliminate tau-induced elevation of reactive oxygen species. Oxygen 133-139 microtubule associated protein tau Homo sapiens 99-102 7585153-4 1995 We now show that as a consequence of glycation, PHF-tau from AD and AGE-tau generate oxygen free radicals, thereby activating transcription via nuclear factor-kappa B, increasing amyloid beta-protein precursor and release of approximately 4 kD amyloid beta-peptides. Oxygen 85-91 microtubule associated protein tau Homo sapiens 48-55 7585153-4 1995 We now show that as a consequence of glycation, PHF-tau from AD and AGE-tau generate oxygen free radicals, thereby activating transcription via nuclear factor-kappa B, increasing amyloid beta-protein precursor and release of approximately 4 kD amyloid beta-peptides. Oxygen 85-91 microtubule associated protein tau Homo sapiens 52-55 33811560-8 2021 Among KTRs, tau showed a moderate negative correlation with Peak VO2 (rho = - 0.52) and Oxygen uptake efficiency slope (OUES) (r = - 0.57) while no significant correlation with Hb and peak heart rate. Oxygen 88-94 microtubule associated protein tau Homo sapiens 12-15 34140566-3 2021 Our primary objective was to investigate the associations of post-stroke fatigue severity with oxygen uptake ((Formula: see text)O2) at peak exercise and the time constant of (Formula: see text)O2 kinetics (tau(Formula: see text)O2) at exercise onset. Oxygen 194-196 microtubule associated protein tau Homo sapiens 207-210 34140566-3 2021 Our primary objective was to investigate the associations of post-stroke fatigue severity with oxygen uptake ((Formula: see text)O2) at peak exercise and the time constant of (Formula: see text)O2 kinetics (tau(Formula: see text)O2) at exercise onset. Oxygen 229-231 microtubule associated protein tau Homo sapiens 207-210 34140566-7 2021 The FSS score was not associated with (Formula: see text)O2 at peak exercise during a symptom-limited graded exercise test (rho = - 0.264; p = 0.224), whereas it was significantly associated with tau(Formula: see text)O2 during a submaximal constant-load exercise test (rho = 0.530; p = 0.009). Oxygen 218-220 microtubule associated protein tau Homo sapiens 196-199 34168725-5 2021 In this context, the hyperphosphorylation of tau and the formation of neurofibrillary tangles have been associated with the dysfunction of the phosphatidylinositol 3-kinase and mitogen-activated protein kinase pathways in the nervous tissues as well as the decrease in the expression of GLUT-1 and GLUT-3 in the different areas of the brain, increase in reactive oxygen species, and production of mitochondrial alterations that occur in T2DM. Oxygen 363-369 microtubule associated protein tau Homo sapiens 45-48 35504110-6 2022 The Abeta42, T-tau, and P-T181-tau levels in the plasma neuronal-derived exosomes were associated with an increased risk of cognitive impairment in OSA patients after additional adjustment for age, gender, education, vascular risk factors, apnea-hypopnea index (AHI) or oxygen reduction index (ODI). Oxygen 270-276 microtubule associated protein tau Homo sapiens 31-34 33949507-5 2021 The contact time required for vibrational relaxation to take place is tau >= 150 fs for non-reacting and tau >= 330 fs for reacting (oxygen atom exchange) trajectories and the two processes are shown to probe different parts of the global potential energy surface. Oxygen 133-139 microtubule associated protein tau Homo sapiens 70-73 33949507-5 2021 The contact time required for vibrational relaxation to take place is tau >= 150 fs for non-reacting and tau >= 330 fs for reacting (oxygen atom exchange) trajectories and the two processes are shown to probe different parts of the global potential energy surface. Oxygen 133-139 microtubule associated protein tau Homo sapiens 105-108 33657673-4 2021 We found that a diluted aqueous mixture of Tau-NHCl and H2 O2 acts as a slow and long-lasting potential source of 1 O2 . Oxygen 59-61 microtubule associated protein tau Homo sapiens 43-46 33657673-9 2021 We found that Tau-NHCl is more stable and adequate for the production of 1 O2 . Oxygen 75-77 microtubule associated protein tau Homo sapiens 14-17 33054659-1 2020 At the onset of an exercise transition, exponential modeling to calculate a time constant (tau) is the conventional method to analyze pulmonary oxygen uptake (VO2p) kinetics for moderate and heavy exercise. Oxygen 144-150 microtubule associated protein tau Homo sapiens 91-94