PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 17506535-0 2007 Mechanism of OH radical reactions with HCN and CH3CN: OH regeneration in the presence of O2. Oxygen 89-91 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 39-42 17506535-1 2007 A theoretical study on the mechanism of the OH reactions with HCN and CH(3)CN, in the presence of O2, is presented. Oxygen 98-100 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 62-65 14714198-2 2004 ACC is oxidized at the expense of O(2) to yield ethylene, HCN, CO(2), and two waters. Oxygen 34-38 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 58-61 16366623-6 2005 The complexes have an L-shaped structure with a hydrogen bond between the oxygen atom of ketene and the hydrogen atom of HCN or HNC. Oxygen 74-80 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 121-124 33750458-0 2021 LncRNA MALAT1 aggravates oxygen-glucose deprivation/reoxygenation-induced neuronal endoplasmic reticulum stress and apoptosis via the miR-195a-5p/HMGA1 axis. Oxygen 25-31 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 7-13 33750458-2 2021 RESULTS: Using an oxygen-glucose deprivation/reoxygenation (OGD/R) cell model, we determined that the expression of MALAT1 was significantly increased during OGD/R. Oxygen 18-24 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 116-122 32691071-0 2020 Retraction: LncRNA MALAT1up-regulates VEGF-A and ANGPT2 to promote angiogenesis in brain microvascular endothelial cells against oxygen-glucose deprivation via targeting miR-145. Oxygen 129-135 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 19-25 32790302-5 2020 HCN was proven to be oxidized by lattice oxygen of the catalyst to CO2 and NO which enters into the NH3-SCR reaction. Oxygen 41-47 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 0-3 33519668-11 2020 Both MALAT1 downregulation and miR-375 upregulation reversed the inhibitory effect of oxygen and glucose deprivation (OGD)/R on cell viability and the promoting effects on LDH level, cell apoptosis, and inflammatory factors levels. Oxygen 86-92 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 5-11 31823095-0 2019 LncRNA MALAT1 Promotes Oxygen-Glucose Deprivation and Reoxygenation Induced Cardiomyocytes Injury Through Sponging miR-20b to Enhance beclin1-Mediated Autophagy. Oxygen 23-29 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 7-13 32579210-0 2020 Expression of Concern: LncRNA MALAT1 up-regulates VEGF-A and ANGPT2 to promote angiogenesis in brain microvascular endothelial cells against oxygen-glucose deprivation via targetting miR-145. Oxygen 141-147 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 30-36 32401514-5 2020 Was there any impact of atomic oxygen in production of early atmospheric HCN for the emergence of life? Oxygen 31-37 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 73-76 32401514-9 2020 We propose that addition reactions of atomic oxygen with CH3NO and CH2NO might act as potential source for early atmospheric HCN. Oxygen 45-51 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 126-129 30301563-0 2019 Hyperbaric oxygen boosts long noncoding RNA MALAT1 exosome secretion to suppress microRNA-92a expression in therapeutic angiogenesis. Oxygen 11-17 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 44-50 27499160-3 2016 Since Malat1 was recently shown to be upregulated during hypoxia, the objective of this study was to determine the contribution of AMPK in the mechanistic pathways regulating Malat1 expression in low oxygen conditions. Oxygen 200-206 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 175-181 29065330-7 2018 These results also demonstrated the notable influence of air, oxygen, and the saline medium in HCN polymer formation. Oxygen 62-68 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 95-98 28880555-1 2017 The proton affinity (PA) on the oxygen atom in silanol and siloxane derivatives is enhanced by the formation of tetrel bonds with small Lewis bases [B R3SiOH, B R3SiOSiR3, B R3SiOSiR3 B; B = H2O, CO, NH3, HCN, H2S; R = H, Me], as shown by MP2/jul-cc-pVTZ calculations. Oxygen 32-38 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 213-216 27499160-4 2016 Compared to those cultured in 21% O2 conditions, HeLa cells incubated in 1.5% O2 expressed more Malat1 transcripts. Oxygen 78-80 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 96-102 27499160-6 2016 Interestingly, pharmacological stimulation of AMPK increased Malat1 promoter transactivation in 21% O2 conditions, whereas inhibition of either AMPK or its upstream activator CaMKK completely abolished the augmentation of Malat1 under hypoxia. Oxygen 100-102 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 61-67 27499160-8 2016 Overexpression of hypoxia-inducible factor-1alpha (HIF-1alpha) increased Malat1 expression in 21% O2 conditions, whereas pharmacological inhibition of HIF-1alpha blocked the impact of hypoxia on the Malat1 promoter. Oxygen 98-100 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 73-79 25313472-2 2015 The specific hypothesis is that PPA can be readily grafted onto the face surfaces of exfoliated HCN, which has reactive apical oxygen atoms. Oxygen 127-133 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 96-99 24733837-3 2014 Here, we show that hyperpolarization-activated, cAMP-gated (HCN) channels--which conduct the hyperpolarization-activated current (Ih) and the voltage-insensitive instantaneous current (Iinst), and contribute to diverse physiological functions including learning and memory, cardiac pacemaking, and the sensation of pain--are subject to modification by (1)O2. Oxygen 355-357 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 60-63 24733837-6 2014 The modification of HCN channel function is a photodynamic process that involves (1)O2, as supported by the dependence on dissolved oxygen in solutions, the inhibitory effect by a (1)O2 scavenger, and the results with the HCN2-SOG fusion protein. Oxygen 81-86 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 20-23 24733837-6 2014 The modification of HCN channel function is a photodynamic process that involves (1)O2, as supported by the dependence on dissolved oxygen in solutions, the inhibitory effect by a (1)O2 scavenger, and the results with the HCN2-SOG fusion protein. Oxygen 132-138 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 20-23 24733837-6 2014 The modification of HCN channel function is a photodynamic process that involves (1)O2, as supported by the dependence on dissolved oxygen in solutions, the inhibitory effect by a (1)O2 scavenger, and the results with the HCN2-SOG fusion protein. Oxygen 180-185 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 20-23 24733837-8 2014 We identified a histidine residue (H434 in S6) near the activation gate in the pore critical for (1)O2 modulation of HCN function. Oxygen 97-102 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 117-120 24691535-3 2014 The interaction between HCN and rGO with oxygen-containing group can result in the formation of hydrogen bonds, N H and O H. The adsorption of HCN on rGO depends on the type and location of oxygen-containing group in rGO. Oxygen 41-47 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 24-27 24691535-3 2014 The interaction between HCN and rGO with oxygen-containing group can result in the formation of hydrogen bonds, N H and O H. The adsorption of HCN on rGO depends on the type and location of oxygen-containing group in rGO. Oxygen 41-47 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 155-158 24691535-3 2014 The interaction between HCN and rGO with oxygen-containing group can result in the formation of hydrogen bonds, N H and O H. The adsorption of HCN on rGO depends on the type and location of oxygen-containing group in rGO. Oxygen 202-208 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 24-27 24691535-3 2014 The interaction between HCN and rGO with oxygen-containing group can result in the formation of hydrogen bonds, N H and O H. The adsorption of HCN on rGO depends on the type and location of oxygen-containing group in rGO. Oxygen 202-208 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 155-158 24691535-5 2014 The adsorption of HCN is much stronger in rGO with oxygen-containing group on the surface than that at the edge. Oxygen 51-57 metastasis associated lung adenocarcinoma transcript 1 Homo sapiens 18-21