PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 34734081-2 2021 The objective of this study was to analyze the association between the serum S100B protein, the Gosling pulsatility index (PI), and the level of oxygen saturation at the tip of the internal jugular vein (SjVO2%) in patients diagnosed with severe TBI. Oxygen 145-151 S100 calcium binding protein B Homo sapiens 77-82 24366505-0 2014 Changes in cerebral oxygen saturation correlate with S100B in infants undergoing cardiac surgery with cardiopulmonary bypass. Oxygen 20-26 S100 calcium binding protein B Homo sapiens 53-58 25251247-0 2014 Nef reaction with molecular oxygen in the absence of metal additives, and mechanistic insights. Oxygen 28-34 S100 calcium binding protein B Homo sapiens 0-3 25251247-1 2014 A Nef reaction has been developed that is conducted under mildly basic conditions with molecular oxygen as an oxidant, without the need for metal additives. Oxygen 97-103 S100 calcium binding protein B Homo sapiens 2-5 25251247-2 2014 Whereas nitroalkanes are converted into ketones in good yield, nitroalkenes are transformed into alpha,beta-unsaturated ketones in one-pot by double-bond isomerization followed by the oxygen-mediated Nef reaction. Oxygen 184-190 S100 calcium binding protein B Homo sapiens 200-203 31794767-6 2020 S100 proteins are released from monocytes, smooth muscle cells and endothelial cells in response to cellular stress stimuli, and then the binding of S100 proteins to RAGE activate downstream signaling such as transcription factor kappa B (NF-kappaB) translocation and reactive oxygen species (ROS) production, which act as a positive feedback loop for inducing pro-inflammatory phenotype in a wide variety of cell types including endothelial cells, vascular smooth muscle cells and leukocytes. Oxygen 277-283 S100 calcium binding protein B Homo sapiens 0-4 31794767-6 2020 S100 proteins are released from monocytes, smooth muscle cells and endothelial cells in response to cellular stress stimuli, and then the binding of S100 proteins to RAGE activate downstream signaling such as transcription factor kappa B (NF-kappaB) translocation and reactive oxygen species (ROS) production, which act as a positive feedback loop for inducing pro-inflammatory phenotype in a wide variety of cell types including endothelial cells, vascular smooth muscle cells and leukocytes. Oxygen 277-283 S100 calcium binding protein B Homo sapiens 149-153 24366505-13 2014 Although the absolute cerebral regional saturation on cardiopulmonary bypass was not associated with S100B elevation, patients who had arterial-cerebral oxygen saturation difference greater than 50 at any time during cardiopulmonary bypass had a higher S100B peak (mean +- SE: 1.053 +- 0.080 vs 0.504 +- 0.039 ng/mL; p < 0.0001). Oxygen 153-159 S100 calcium binding protein B Homo sapiens 253-258 15745143-12 2005 The inverse relationship of both cerebral and renal markers with DO2 and VO2 suggests that increased levels of S100beta and NAG during CPB may primarily be caused by an oxygen deficit and secondary to the inflammatory response. Oxygen 169-175 S100 calcium binding protein B Homo sapiens 111-119 23989819-15 2013 The low S100beta variance during the fluid restrictive MCABG technique may be due to more efficient oxygen transport to the brain provided by significantly higher perioperative Ht levels. Oxygen 100-106 S100 calcium binding protein B Homo sapiens 8-16 17984939-10 2007 The difference between the evening and morning S100B levels correlated negatively with AHI and ODI and positively with basal saturation and average minimal oxygen saturation. Oxygen 156-162 S100 calcium binding protein B Homo sapiens 47-52 17891653-10 2007 A significant correlation was found between arterial oxygen partial pressure and individual S100B concentration in the pulmonary and systemic bloodstream in the entire study group (R = -0.66 and R = 0.71, respectively; p < 0.05, for both). Oxygen 53-59 S100 calcium binding protein B Homo sapiens 92-97 15745143-10 2005 After protamine infusion, total body oxygen delivery and consumption correlated negatively with S100beta levels (both p < 0.05) and with NAG levels (both p < 0.01). Oxygen 37-43 S100 calcium binding protein B Homo sapiens 96-104 22360297-3 2012 This study evaluates the association between S100beta, neuron-specific enolase (NSE), and glial fibrillary acidic protein (GFAP), detected in the serum of severe TBI patients and CH as measured by brain tissue oxygen partial pressure (Pbo(2)). Oxygen 210-216 S100 calcium binding protein B Homo sapiens 45-53 17923015-11 2007 HES130/0.4 lowers the S100B protein levels from the beginning of CPB to one hour after the termination of CPB with the probable mechanism of improving the cerebral metabolism of oxygen. Oxygen 178-184 S100 calcium binding protein B Homo sapiens 22-27 16782283-5 2006 While oxygen, serum and glucose deprivation triggered the most rapid release of S100B, serum and glucose deprivation provoked comparable levels of released S100B at the later time points. Oxygen 6-12 S100 calcium binding protein B Homo sapiens 80-85 16782283-8 2006 Interestingly, S100B mRNA expression was potently downregulated after 12 and 24 h of oxygen, serum and glucose deprivation, and prolonged oxygen, serum and glucose deprivation for 48 h was associated with a significant reduction of S100B release at later time intervals, whereas lactate dehydrogenase levels remained constant. Oxygen 85-91 S100 calcium binding protein B Homo sapiens 15-20 16782283-8 2006 Interestingly, S100B mRNA expression was potently downregulated after 12 and 24 h of oxygen, serum and glucose deprivation, and prolonged oxygen, serum and glucose deprivation for 48 h was associated with a significant reduction of S100B release at later time intervals, whereas lactate dehydrogenase levels remained constant. Oxygen 138-144 S100 calcium binding protein B Homo sapiens 15-20 16782283-8 2006 Interestingly, S100B mRNA expression was potently downregulated after 12 and 24 h of oxygen, serum and glucose deprivation, and prolonged oxygen, serum and glucose deprivation for 48 h was associated with a significant reduction of S100B release at later time intervals, whereas lactate dehydrogenase levels remained constant. Oxygen 138-144 S100 calcium binding protein B Homo sapiens 232-237 12505746-0 2003 The potential value of the protein S-100B level as a criterion for hyperbaric oxygen treatment and prognostic marker in carbon monoxide poisoned patients. Oxygen 78-84 S100 calcium binding protein B Homo sapiens 27-41