PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24788249-8	2014	Furthermore, iodide efflux assays confirmed that NHERF1 and P-ERM are necessary for VIP regulation of the stability and sustained activity of membrane CFTR.	Iodides	13-19	vasoactive intestinal peptide	Homo sapiens	84-87	
14744818-7	2004	We found that VIP (EC(50) approximately 7.6 nM) and PACAP-27 (EC(50) approximately 10 nM) stimulated glibenclamide-sensitive and DIDS-insensitive iodide efflux in Calu-3 cells.	Iodides	146-152	vasoactive intestinal peptide	Homo sapiens	14-17	
21411725-4	2011	Iodide effluxes were used to monitor the presence of VIP-rescued functional F508del-CFTR channels at the surface of JME/CF15 cells maintained at 37 C. Iodide efflux peaks measured in response to stimulation with forskolin were insensitive to PKC alpha, beta, gamma, delta, zeta inhibitors.	Iodides	151-157	vasoactive intestinal peptide	Homo sapiens	53-56	
21411725-4	2011	Iodide effluxes were used to monitor the presence of VIP-rescued functional F508del-CFTR channels at the surface of JME/CF15 cells maintained at 37 C. Iodide efflux peaks measured in response to stimulation with forskolin were insensitive to PKC alpha, beta, gamma, delta, zeta inhibitors.	Iodides	0-6	vasoactive intestinal peptide	Homo sapiens	53-56	
19584307-5	2009	One hour of treatment with VIP strongly increased F508del-CFTR activity, with iodide efflux peaks three times higher than with untreated cells.	Iodides	78-84	vasoactive intestinal peptide	Homo sapiens	27-30	
19584307-6	2009	At 37 degrees C, VIP-treated cells, but not untreated controls, showed significant iodide efflux peaks that were sensitive to the CFTR inhibitor 3-[(3-trifluoromethyl)phenyl]-5-[(4-carboxyphenyl)methylene]-2-thioxo-4-thiazolidinone (CFTR(inh)-172).	Iodides	83-89	vasoactive intestinal peptide	Homo sapiens	17-20