PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26660892-3	2016	Iodide plays an important role in thyroid function at multiple steps of thyroid colloid synthesis and transport among which sodium/iodide symporter (NIS) and pendrin are essential.	Iodides	0-6	solute carrier family 26, member 4	Mus musculus	158-165	
30333321-4	2018	SLC26A7 is a member of the same transporter family as SLC26A4 (pendrin), an anion exchanger with affinity for iodide and chloride (among others), whose gene mutations cause congenital deafness and dyshormonogenic goiter.	Iodides	110-116	solute carrier family 26, member 4	Mus musculus	54-61	
30333321-4	2018	SLC26A7 is a member of the same transporter family as SLC26A4 (pendrin), an anion exchanger with affinity for iodide and chloride (among others), whose gene mutations cause congenital deafness and dyshormonogenic goiter.	Iodides	110-116	solute carrier family 26, member 4	Mus musculus	63-70	
26660892-9	2016	Based on these findings, we concluded that the occurrence of thyroid colloid retention exacerbated by excess iodide was associated with the suppression of NIS and pendrin expression, providing an additional insight of the potential mechanism of action of excess iodide on thyroid gland.	Iodides	109-115	solute carrier family 26, member 4	Mus musculus	163-170	
26660892-9	2016	Based on these findings, we concluded that the occurrence of thyroid colloid retention exacerbated by excess iodide was associated with the suppression of NIS and pendrin expression, providing an additional insight of the potential mechanism of action of excess iodide on thyroid gland.	Iodides	262-268	solute carrier family 26, member 4	Mus musculus	163-170	
17055311-1	2006	Pendrin is a membrane transport protein which functions as the transporter of chloride, bicarbonate, formate, and iodide.	Iodides	114-120	solute carrier family 26, member 4	Mus musculus	0-7	
24429827-1	2013	BACKGROUND: The anion exchanger pendrin (SLC26A4) is required for proper development of the inner ear, and contributes to iodide organification in thyroid glands as well as anion transport in various epithelia, such as airways and renal tubules.	Iodides	122-128	solute carrier family 26, member 4	Mus musculus	32-39	
24429827-1	2013	BACKGROUND: The anion exchanger pendrin (SLC26A4) is required for proper development of the inner ear, and contributes to iodide organification in thyroid glands as well as anion transport in various epithelia, such as airways and renal tubules.	Iodides	122-128	solute carrier family 26, member 4	Mus musculus	41-48	
22116363-1	2011	The anion exchanger pendrin (Pds, SLC26A4) transports various anions including bicarbonate, chloride and iodide.	Iodides	105-111	solute carrier family 26, member 4	Mus musculus	20-27	
22116363-1	2011	The anion exchanger pendrin (Pds, SLC26A4) transports various anions including bicarbonate, chloride and iodide.	Iodides	105-111	solute carrier family 26, member 4	Mus musculus	34-41	
20834201-3	2011	Since pendrin can transport iodide in vitro, variations in iodide supply have been claimed to account for the thyroid phenotype associated with pendrin defects.	Iodides	28-34	solute carrier family 26, member 4	Mus musculus	6-13	
20834201-3	2011	Since pendrin can transport iodide in vitro, variations in iodide supply have been claimed to account for the thyroid phenotype associated with pendrin defects.	Iodides	59-65	solute carrier family 26, member 4	Mus musculus	144-151	
20834201-4	2011	AIM: The Slc26a4 (-/-) mouse model was used to test the hypothesis that iodide supply may influence the penetrance and expressivity of SLC26A4 mutations.	Iodides	72-78	solute carrier family 26, member 4	Mus musculus	135-142	
22116370-1	2011	SLC26A4 encodes pendrin, a transporter exchanging anions such as chloride, bicarbonate, and iodide.	Iodides	92-98	solute carrier family 26, member 4	Mus musculus	0-7	
22116370-1	2011	SLC26A4 encodes pendrin, a transporter exchanging anions such as chloride, bicarbonate, and iodide.	Iodides	92-98	solute carrier family 26, member 4	Mus musculus	16-23	
19605545-0	2009	Role of pendrin in iodide balance: going with the flow.	Iodides	19-25	solute carrier family 26, member 4	Mus musculus	8-15	
16306076-10	2006	Expression of pendrin, an I-/Cl- exchanger involved in apical iodide efflux, was selectively decreased by 60% in KO mice at mRNA and protein levels.	Iodides	62-68	solute carrier family 26, member 4	Mus musculus	14-21	
12388138-3	2003	We now report the existence of a second iodide transporter, pendrin, which is also essential for iodide accumulation in milk.	Iodides	40-46	solute carrier family 26, member 4	Mus musculus	60-67	
12388138-6	2003	The prolactin effect on iodide uptake into cultured mammary tissues was abolished by pendrin transport inhibitors, including DIDS, furosemide, and probenecid.	Iodides	24-30	solute carrier family 26, member 4	Mus musculus	85-92	
12388138-7	2003	These studies suggest that the prolactin stimulation of pendrin activity is an essential element in the prolactin stimulation of iodide uptake into milk.	Iodides	129-135	solute carrier family 26, member 4	Mus musculus	56-63