PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
21035537-6	2011	Iodide tunes down the biological activity of autonomous thyrocytes and may thus be of therapeutic benefit not only to prevent the occurrence of somatic TSHR mutations, causing thyroid autonomy, but also to slow down the development of clinically relevant disease.	Iodides	0-6	thyroid stimulating hormone receptor	Rattus norvegicus	152-156	
15691889-3	2005	We aimed to investigate whether iodide could modulate the biological effects of activating TSHR mutations.	Iodides	32-38	thyroid stimulating hormone receptor	Rattus norvegicus	91-95	
18272324-5	2008	We also showed that iodide excess inhibits the expression of essential genes for thyroid differentiation: Tshr, Nis, Tg, and Tpo.	Iodides	20-26	thyroid stimulating hormone receptor	Rattus norvegicus	106-110	
15691889-13	2005	In conclusion, our studies indicate that the biological effects of activating TSHR mutations vary with the ambient iodide supply and could be masked in regions of high iodine intake.	Iodides	115-121	thyroid stimulating hormone receptor	Rattus norvegicus	78-82	
7850004-0	1995	Augmentation of thyrotropin-induced iodide uptake in FRTL-5 cells by immunoglobulins containing blocking thyrotropin-receptor antibodies under NaCl-depleted conditions.	Iodides	36-42	thyroid stimulating hormone receptor	Rattus norvegicus	105-125