PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22406437-2	2012	This study investigated a role for the glutathione S-transferase (Gst) isoforms alpha (a), mu (m) and pi (p) and the transcription factors, Ahr and Nrf2, during DMBA-induced ovotoxicity, and their regulation by phosphatidylinositol-3 kinase (PI3K) signaling.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	161-165	aryl hydrocarbon receptor	Rattus norvegicus	140-143	
34867043-4	2021	Therefore, the main aims of this study are to examine the effect of MET on protecting against rat breast carcinogenesis induced by DMBA and to explore whether this effect is medicated through the AhR pathway.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	131-135	aryl hydrocarbon receptor	Rattus norvegicus	196-199	
22406437-8	2012	Ahr mRNA and protein increased after 2 and 4 days of DMBA exposure, respectively and DMBA increased NRF2 protein level after 4 days.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	53-57	aryl hydrocarbon receptor	Rattus norvegicus	0-3	
15990133-4	2006	We investigated the hepatic expression of CYP1 and AhR following oral administration of DMBA (100 mg/kg b.w.)	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	88-92	aryl hydrocarbon receptor	Rattus norvegicus	51-54	
11097088-11	2000	While both AhR-regulated CYP1A1 and CYP1B1 mRNAs were induced in breast tissue within 6 h of DMBA gavage, only CYP1B1 mRNA remained elevated in tumors.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	93-97	aryl hydrocarbon receptor	Rattus norvegicus	11-14