PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27461625-6	2016	RESULTS: The treatment of DMBA-exposed rats with ZOL and RT, both alone and in combination, successfully upregulates the transcriptional levels of Bax, caspase-3, caspase-9, p21, and BRCA 1 in mammary tissues, which may account for the elevated apoptotic activities observed and the eventual inhibition of tumor growth.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	26-30	KRAS proto-oncogene, GTPase	Rattus norvegicus	174-177	
12713563-0	2003	Overexpression of p21, cyclin E and decreased expression of p27 in DMBA (7, 12-dimethylbenzanthracene)-induced rat ovarian carcinogenesis.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	67-71	KRAS proto-oncogene, GTPase	Rattus norvegicus	18-21	
12713563-10	2003	These results suggest that the increased expression of cyclin E and p21, and the decreased expression of p27, occur in DMBA-induced rat ovarian carcinogenesis and result in tumor progression.	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	119-123	KRAS proto-oncogene, GTPase	Rattus norvegicus	68-71	
1919181-1	1991	In the present study, ras oncogene product p21 was analyzed immunohistochemically in rat ovarian tumors induced by 7,12 dimethylbenz (a) anthracene (DMBA).	6,11-dimethylbenzo(b)naphtho(2,3-d)thiophene	149-153	KRAS proto-oncogene, GTPase	Rattus norvegicus	43-46