PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26880135-7	2016	Studies have also demonstrated a key role of cytokines such as stromal cell-derived factor-1, tumor necrosis factor-alpha, as well as vascular endothelial growth factor, in regulating the migration of BMCs.	bmcs	201-205	C-X-C motif chemokine ligand 12	Homo sapiens	63-121	
30261919-8	2018	RESULTS: We showed that Sdf-1 treatment increased the number of CXCR4+BMCs able to bind the myofiber and occupy the satellite cell niche.	bmcs	70-74	C-X-C motif chemokine ligand 12	Homo sapiens	24-29	
30261919-10	2018	CXCR4+BMCs, pretreated by the coculture with myofibers and Sdf-1, participated in myotube formation in vitro and also myofiber reconstruction in vivo.	bmcs	6-10	C-X-C motif chemokine ligand 12	Homo sapiens	59-64	
30261919-11	2018	We also showed that Sdf-1 overexpression in vivo (in injured and regenerating muscles) supported the participation of CXCR4+BMCs in new myofiber formation.	bmcs	124-128	C-X-C motif chemokine ligand 12	Homo sapiens	20-25	
25047264-8	2014	mRNA levels of SDF-1 was increased whereas its major inhibitor dipeptidylpeptidase IV (DPP IV) is decreased in PA, suggesting that the SDF-1 axis plays a role in the migration of BMCs into PA.	bmcs	179-183	C-X-C motif chemokine ligand 12	Homo sapiens	15-20	
25047264-8	2014	mRNA levels of SDF-1 was increased whereas its major inhibitor dipeptidylpeptidase IV (DPP IV) is decreased in PA, suggesting that the SDF-1 axis plays a role in the migration of BMCs into PA.	bmcs	179-183	C-X-C motif chemokine ligand 12	Homo sapiens	135-140