PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
14980935-4	2004	HES was found to inhibit lung neutrophil accumulation, cytokine-induced neutrophil chemoattractant protein, and NF-kappaB activation in parallel and to inhibit CD11b expression in a dose-dependent manner.	Hydroxyethyl Starch Derivatives	0-3	nuclear factor kappa B subunit 1	Homo sapiens	112-121	
14980935-5	2004	These findings demonstrate that HES has beneficial effects on capillary leak in acute lung injury and that the mechanisms underlying this action involve an antiinflammatory effect of HES, including inhibition of NF-kappaB activation.	Hydroxyethyl Starch Derivatives	32-35	nuclear factor kappa B subunit 1	Homo sapiens	212-221	
14980935-5	2004	These findings demonstrate that HES has beneficial effects on capillary leak in acute lung injury and that the mechanisms underlying this action involve an antiinflammatory effect of HES, including inhibition of NF-kappaB activation.	Hydroxyethyl Starch Derivatives	183-186	nuclear factor kappa B subunit 1	Homo sapiens	212-221	
14584760-2	2003	The present study tested the hypothesis that HES has effects on nuclear factor kappa B (NF-kappaB) activation and the expression of inflammatory mediators induced by lipopolysaccharide.	Hydroxyethyl Starch Derivatives	45-48	nuclear factor kappa B subunit 1	Homo sapiens	88-97	
14584760-6	2003	Treatment of rats with HES (3.75 and 7.5 ml/kg) prevented LPS-induced NF-kappaB activation, and inhibited, in a dose-related manner, LPS-induced TNF-alpha and CINC expression.	Hydroxyethyl Starch Derivatives	23-26	nuclear factor kappa B subunit 1	Homo sapiens	70-79