PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31448845-5	2020	Although a single recessive variant in CRAT has been recently associated with neurodegeneration with brain iron accumulation, our study reports the first kinetic analysis of naturally occurring CRAT variants and demonstrates the genetic basis of carnitine acetyltransferase deficiency in a case of mitochondrial encephalopathy.	Iron	107-111	carnitine O-acetyltransferase	Homo sapiens	39-43	
31448845-5	2020	Although a single recessive variant in CRAT has been recently associated with neurodegeneration with brain iron accumulation, our study reports the first kinetic analysis of naturally occurring CRAT variants and demonstrates the genetic basis of carnitine acetyltransferase deficiency in a case of mitochondrial encephalopathy.	Iron	107-111	carnitine O-acetyltransferase	Homo sapiens	194-198	
29395073-3	2018	After studying fibroblast cell lines from subjects carrying both known and unreported biallelic mutations in CRAT and REPS1, we ascribe iron overload to the abnormal recycling of transferrin receptor (TfR1) and the reduction of TfR1 palmitoylation in NBIA.	Iron	136-140	carnitine O-acetyltransferase	Homo sapiens	109-113