PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24503166-8	2014	In addition, CPA inhibited inducible nitric oxide synthase (iNOS), activation of nuclear factor (NF)-kappaB by degradation and phosphorylation of IkappaBalpha, and attenuated phosphorylation of mitogen-activated protein kinases (MAPKs; ERK 1/2, p38 and JNK) from mice challenged with LPS.	cpa	13-16	mitogen-activated protein kinase 3	Mus musculus	236-243	
20930592-4	2010	In wild-type hearts, CPA (100 nM) significantly (P < 0.05) improved contractility (52.7 +- 6.2% versus 23.9 +- 4.9% of preischemia), left ventricular developed pressure, end diastolic pressure; reduced infarct size (7.9 +- 1.7% versus 23.9 +- 6.6% area at risk); decreased lactate dehydrogenase efflux; and increased ERK1/2 phosphorylation at 60 minutes of reperfusion.	cpa	21-24	mitogen-activated protein kinase 3	Mus musculus	320-326