PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24597568-6	2015	KEY RESULTS: The extinction of CPA behaviour increased the phosphorylation of ERK and CREB in the dorsal hippocampus (DH) and basolateral amygdala (BLA), but not in the central amygdala (CeA).	cpa	31-34	Eph receptor B1	Rattus norvegicus	78-81	
24597568-9	2015	CONCLUSIONS AND IMPLICATIONS: These results suggest that activation of ERK and CREB in the BLA and DH is involved in the extinction of CPA behaviour and that the DH, via a direct or indirect pathway, modulates the activity of ERK and CREB in the BLA through activation of NMDA receptors after extinction training.	cpa	135-138	Eph receptor B1	Rattus norvegicus	71-74	
28732667-7	2017	Importantly, we found that administration of exogenous BDNF into the rACC of intact rats was sufficient to produce CPA, while selectively blocking phosphorylated extracellular signal regulated kinase (p-ERK) with a mitogen-activated protein kinase (MAPK) inhibitor U0126 completely abolished the acquisition of BDNF-induced CPA.	cpa	324-327	Eph receptor B1	Rattus norvegicus	203-206	
16707501-3	2006	In cultured adult rat ventricular myocytes (ARVM), the A(1) receptor agonist cyclopentyladenosine (CPA) inhibited the increase in NHE1 phosphorylation induced by the alpha(1)-adrenoreceptor agonist phenylephrine, without affecting activation of the ERK/RSK pathway.	cpa	99-102	Eph receptor B1	Rattus norvegicus	249-252	
14622107-8	2003	cPA-elicited ERK activation was blocked by K252a and PKI.	cpa	0-3	Eph receptor B1	Rattus norvegicus	13-16