PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28950822-7	2017	Statins also enhance the expression, enzymatic activity and coupling of endothelial nitric oxide synthase (eNOS), through mevalonate-dependent effects.	Mevalonic Acid	122-132	nitric oxide synthase 3	Homo sapiens	72-105	
11079678-13	2000	CONCLUSIONS: In humans, SVEC inhibition of HMG-CoA/mevalonate pathway contributes to the enhanced eNOS expression and NO release by cerivastatin, whereas in SMC, inhibition of this pathway only partially explains cerivastatin-induced cell growth arrest.	Mevalonic Acid	51-61	nitric oxide synthase 3	Homo sapiens	98-102	
32382091-9	2020	Pharmacological inhibitors of the mevalonate pathway prevented fractionated-radiation-induced suppression of KLF2, TM, and eNOS expression.	Mevalonic Acid	34-44	nitric oxide synthase 3	Homo sapiens	123-127	
16165089-5	2005	The effects on eNOS expression mediated through rosuvastatin could be reversed by treatment with mevalonate indicating inhibition of HMG-CoA reductase as the underlying mechanism.	Mevalonic Acid	97-107	nitric oxide synthase 3	Homo sapiens	15-19	
15049129-7	2004	In addition, the expression of endothelial nitric oxide synthase was strongly induced by Pitavastatin, and was suppressed by mevalonic acid and geranylgeranyl pyrophosphate by Western blot analysis.	Mevalonic Acid	125-139	nitric oxide synthase 3	Homo sapiens	31-64