PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 21729327-5 2011 Moreover, our in vitro studies showed that Abeta1-42 and DNA-damaging drugs, namely, etoposide and homocysteine, increased the expression ATBF1 level in primary rat cortical neurons, whereas the knockdown of ATBF1 in these neurons protected against neuronal death induced by Abeta1-42, etoposide, and homocysteine, indicating that ATBF1 mediates neuronal death in response to these substances. Etoposide 85-94 zinc finger homeobox 3 Rattus norvegicus 138-143 21729327-5 2011 Moreover, our in vitro studies showed that Abeta1-42 and DNA-damaging drugs, namely, etoposide and homocysteine, increased the expression ATBF1 level in primary rat cortical neurons, whereas the knockdown of ATBF1 in these neurons protected against neuronal death induced by Abeta1-42, etoposide, and homocysteine, indicating that ATBF1 mediates neuronal death in response to these substances. Etoposide 85-94 zinc finger homeobox 3 Rattus norvegicus 208-213 21729327-5 2011 Moreover, our in vitro studies showed that Abeta1-42 and DNA-damaging drugs, namely, etoposide and homocysteine, increased the expression ATBF1 level in primary rat cortical neurons, whereas the knockdown of ATBF1 in these neurons protected against neuronal death induced by Abeta1-42, etoposide, and homocysteine, indicating that ATBF1 mediates neuronal death in response to these substances. Etoposide 85-94 zinc finger homeobox 3 Rattus norvegicus 208-213 21729327-5 2011 Moreover, our in vitro studies showed that Abeta1-42 and DNA-damaging drugs, namely, etoposide and homocysteine, increased the expression ATBF1 level in primary rat cortical neurons, whereas the knockdown of ATBF1 in these neurons protected against neuronal death induced by Abeta1-42, etoposide, and homocysteine, indicating that ATBF1 mediates neuronal death in response to these substances. Etoposide 286-295 zinc finger homeobox 3 Rattus norvegicus 208-213 21729327-5 2011 Moreover, our in vitro studies showed that Abeta1-42 and DNA-damaging drugs, namely, etoposide and homocysteine, increased the expression ATBF1 level in primary rat cortical neurons, whereas the knockdown of ATBF1 in these neurons protected against neuronal death induced by Abeta1-42, etoposide, and homocysteine, indicating that ATBF1 mediates neuronal death in response to these substances. Etoposide 286-295 zinc finger homeobox 3 Rattus norvegicus 208-213