PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29769340-6	2018	HBZ was found to interact with two vital members of the NHEJ core machinery, Ku70 and Ku80, and to be recruited to DSBs in a bZIP-dependent manner in vitro We observed that HBZ expression also resulted in a bZIP-dependent delay in DNA protein kinase (DNA-PK) activation following treatment with etoposide.	Etoposide	295-304	X-ray repair cross complementing 5	Homo sapiens	86-90	
15133299-6	2004	Furthermore, etoposide- and radiosensitive phenotype of xrs-6 cells were corrected by an introduction of the tagged Ku80.	Etoposide	13-22	X-ray repair cross complementing 5	Homo sapiens	116-120	
12384553-0	2002	An antisense oligonucleotide targeted to human Ku86 messenger RNA sensitizes M059K malignant glioma cells to ionizing radiation, bleomycin, and etoposide but not DNA cross-linking agents.	Etoposide	144-153	X-ray repair cross complementing 5	Homo sapiens	47-51	
12384553-5	2002	Moreover, transfection of M059K cells with Ku86 antisense ASOs markedly increased cell death after treatment with ionizing radiation, bleomycin, and etoposide.	Etoposide	149-158	X-ray repair cross complementing 5	Homo sapiens	43-47	
12384553-7	2002	As expected, transfection of M059J cells with Ku86 antisense ASOs did not result in any sensitization to ionizing radiation, bleomycin, or DNA cross-linking agents, but there was a 2-fold increase in sensitivity to etoposide.	Etoposide	215-224	X-ray repair cross complementing 5	Homo sapiens	46-50	
34817176-7	2021	We further demonstrate that myrocins disrupt the interaction of XRCC5 with DNA leading to sensitization of cancer cells to the chemotherapeutic agent etoposide as well as UV-light-induced DNA damage.	Etoposide	150-159	X-ray repair cross complementing 5	Homo sapiens	64-69	
31473257-4	2019	In this research, we found that knockdown of JMJD8 in cancer cells significantly increased cell proliferation, and attenuated ionizing irradiation or etoposide treatment-induced DNA double-strand breaks (DSBs) level through enhancing the expression of Ku70 and Ku80 which are key participants in the non-homologous end-joining repair of DSBs.	Etoposide	150-159	X-ray repair cross complementing 5	Homo sapiens	261-265	
29065392-4	2017	Here we report that Ku80 can be cleaved by caspases-2 at D726 upon a transient etoposide treatment.	Etoposide	79-88	X-ray repair cross complementing 5	Homo sapiens	20-24