PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25607114-5	2015	However, co-treatment with anti-VEGF significantly restored etoposide-induced cell apoptosis and cell cycle arrest, as indicated by the elimination of B-cell lymphoma 2 (Bcl-2), procaspase 3, cyclin B1 and Cdc2.	Etoposide	60-69	cyclin B1	Homo sapiens	192-201	
8895510-2	1996	Exposure of HeLa cells to camptothecin, etoposide or nitrogen mustard for 1 h in S phase resulted in delayed expression of cyclin B1 mRNA during the G2 arrest.	Etoposide	40-49	cyclin B1	Homo sapiens	123-132	
8895510-5	1996	However, with camptothecin or etoposide treatment, while the accumulation of cyclin B1 protein was delayed, its levels eventually surpassed peak levels seen in control cells, in spite of the fact that cells were still blocked in G2.	Etoposide	30-39	cyclin B1	Homo sapiens	77-86	
7812949-6	1995	The same transient activation and subsequent inactivation of cyclin B1/Cdc2 kinase were observed after DNA damage by etoposide or bis-(2-chloroethyl)methylamine hydrochloride.	Etoposide	117-126	cyclin B1	Homo sapiens	61-70	
27297620-0	2016	Activity of cyclin B1 in HL-60 cells treated with etoposide.	Etoposide	50-59	cyclin B1	Homo sapiens	12-21	
27297620-5	2016	Etoposide caused decrease in cell viability, induced apoptosis and G2/M arrest accompanied by enhanced expression of cyclin B1.	Etoposide	0-9	cyclin B1	Homo sapiens	117-126	
27297620-6	2016	Changes in expression and localization of cyclin B1 may constitute a part of the mechanism responsible for resistance of HL-60 cells to etoposide.	Etoposide	136-145	cyclin B1	Homo sapiens	42-51	
12569391-4	2003	The increased sensitivity to etoposide-induced G(2)-M arrest was associated with disruption of cell cycle-related events, including hyperphosphorylation of p34(cdc2) kinase, change in cdc25C expression and phosphorylation, and alteration in cyclin B1 expression.	Etoposide	29-38	cyclin B1	Homo sapiens	241-250	
11911275-6	2001	MG-132 was found to be more effective in combination with drugs such as doxorubicin and etoposide that act in the S/G2-phase of the cell cycle via a mechanism that involves stabilization of cyclin B1 and increased expression of Bax.	Etoposide	88-97	cyclin B1	Homo sapiens	190-199	
11162602-4	2001	The reduction in cyclin B1 expression and G2 arrest were also seen after treatment with etoposide and irinotecan.	Etoposide	88-97	cyclin B1	Homo sapiens	17-26