PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10774745-4	2000	They were also slightly more resistant than the parental cell (AML-2/WT) to etoposide, camptothecin and daunorubicin.	Etoposide	76-85	RUNX family transcription factor 3	Homo sapiens	63-68	
21558800-6	2011	Compared to AML3 cells, AML2 cells are more sensitive to the treatment of the DNA-damaging compounds (doxorubicin and etoposide) and a specific p53-inducing compound (nutlin-3).	Etoposide	118-127	RUNX family transcription factor 3	Homo sapiens	24-28