PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 22495427-0 2012 Polymorphism of genes involved in purine metabolism (XDH, AOX1, MOCOS) in kidney transplant recipients receiving azathioprine. Azathioprine 113-125 xanthine dehydrogenase Homo sapiens 53-56 22495427-2 2012 The aim of this study was to investigate single nucleotide polymorphisms (SNPs) in XDH, AOX1, and MOCOS genes in relation to clinical parameters and risk of drug side effects in a cohort of kidney transplant recipients treated with azathioprine (AZA) as a part of standard immunosuppressive regimen. Azathioprine 232-244 xanthine dehydrogenase Homo sapiens 83-86 22495427-2 2012 The aim of this study was to investigate single nucleotide polymorphisms (SNPs) in XDH, AOX1, and MOCOS genes in relation to clinical parameters and risk of drug side effects in a cohort of kidney transplant recipients treated with azathioprine (AZA) as a part of standard immunosuppressive regimen. Azathioprine 246-249 xanthine dehydrogenase Homo sapiens 83-86 22934314-13 1993 Agents/circumstances to avoid: Azathioprine should be avoided by individuals taking XDH inhibitors. Azathioprine 31-43 xanthine dehydrogenase Homo sapiens 84-87 26863601-2 2016 Although xanthine dehydrogenase (XDH) is the second major contributor to azathioprine breakdown, polymorphisms in XDH have rarely been studied in IBD patients. Azathioprine 73-85 xanthine dehydrogenase Homo sapiens 9-31 26863601-2 2016 Although xanthine dehydrogenase (XDH) is the second major contributor to azathioprine breakdown, polymorphisms in XDH have rarely been studied in IBD patients. Azathioprine 73-85 xanthine dehydrogenase Homo sapiens 33-36 19500084-3 2009 Xanthine oxidase/dehydrogenase (XDH) and aldehyde oxidase (AO) compete with TPMT to inactivate AZA. Azathioprine 95-98 xanthine dehydrogenase Homo sapiens 0-30 19500084-3 2009 Xanthine oxidase/dehydrogenase (XDH) and aldehyde oxidase (AO) compete with TPMT to inactivate AZA. Azathioprine 95-98 xanthine dehydrogenase Homo sapiens 32-35 19500084-4 2009 AIM: To assess whether genetic polymorphism in AOX1, XDH and MOCOS (the product of which activates the essential cofactor for AO and XDH) is associated with AZA treatment outcome in IBD. Azathioprine 157-160 xanthine dehydrogenase Homo sapiens 133-136