PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22495427-0	2012	Polymorphism of genes involved in purine metabolism (XDH, AOX1, MOCOS) in kidney transplant recipients receiving azathioprine.	Azathioprine	113-125	xanthine dehydrogenase	Homo sapiens	53-56	
22495427-2	2012	The aim of this study was to investigate single nucleotide polymorphisms (SNPs) in XDH, AOX1, and MOCOS genes in relation to clinical parameters and risk of drug side effects in a cohort of kidney transplant recipients treated with azathioprine (AZA) as a part of standard immunosuppressive regimen.	Azathioprine	232-244	xanthine dehydrogenase	Homo sapiens	83-86	
22495427-2	2012	The aim of this study was to investigate single nucleotide polymorphisms (SNPs) in XDH, AOX1, and MOCOS genes in relation to clinical parameters and risk of drug side effects in a cohort of kidney transplant recipients treated with azathioprine (AZA) as a part of standard immunosuppressive regimen.	Azathioprine	246-249	xanthine dehydrogenase	Homo sapiens	83-86	
22934314-13	1993	Agents/circumstances to avoid: Azathioprine should be avoided by individuals taking XDH inhibitors.	Azathioprine	31-43	xanthine dehydrogenase	Homo sapiens	84-87	
26863601-2	2016	Although xanthine dehydrogenase (XDH) is the second major contributor to azathioprine breakdown, polymorphisms in XDH have rarely been studied in IBD patients.	Azathioprine	73-85	xanthine dehydrogenase	Homo sapiens	9-31	
26863601-2	2016	Although xanthine dehydrogenase (XDH) is the second major contributor to azathioprine breakdown, polymorphisms in XDH have rarely been studied in IBD patients.	Azathioprine	73-85	xanthine dehydrogenase	Homo sapiens	33-36	
19500084-3	2009	Xanthine oxidase/dehydrogenase (XDH) and aldehyde oxidase (AO) compete with TPMT to inactivate AZA.	Azathioprine	95-98	xanthine dehydrogenase	Homo sapiens	0-30	
19500084-3	2009	Xanthine oxidase/dehydrogenase (XDH) and aldehyde oxidase (AO) compete with TPMT to inactivate AZA.	Azathioprine	95-98	xanthine dehydrogenase	Homo sapiens	32-35	
19500084-4	2009	AIM: To assess whether genetic polymorphism in AOX1, XDH and MOCOS (the product of which activates the essential cofactor for AO and XDH) is associated with AZA treatment outcome in IBD.	Azathioprine	157-160	xanthine dehydrogenase	Homo sapiens	133-136