PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
2135899-8	1990	abolished the vascular and motor responses to exogenous CCK and significantly reduced intestinal hyperemia and increase in VO2 in response to luminal oleic acid but not glucose.	Oleic Acid	150-160	cholecystokinin	Canis lupus familiaris	56-59	
1475410-5	1992	Addition of 10% oleate or acidification of peptone to pH 3.0 profoundly inhibited acid secretion while significantly increasing the pancreatic protein secretion and plasma CCK levels.	Oleic Acid	16-22	cholecystokinin	Canis lupus familiaris	172-175	
1403819-8	1992	Addition to intragastric peptone of 10% oleate or its acidification to pH 3.0 profoundly inhibited the HP secretion and gastrin release but significantly increased pancreatic secretion of protein and plasma levels of CCK and somatostatin.	Oleic Acid	40-46	cholecystokinin	Canis lupus familiaris	217-220	
1403819-16	1992	We conclude that CCK released by a meal of meat, intragastric peptone, oleate or acidified peptone and intravenous bombesin exerts tonic inhibitory influences on gastric acid secretion and that this effect is mediated, at least in part, by somatostatin.	Oleic Acid	71-77	cholecystokinin	Canis lupus familiaris	17-20	
2210484-8	1990	abolished the circulatory, metabolic and secretory responses to CCK and significantly reduced these responses to luminal oleic acid and bile.	Oleic Acid	121-131	cholecystokinin	Canis lupus familiaris	64-67	
111364-11	1979	We have shown that, using our RIA method, there is a measurable rise in plasma CCK LI after intraduodenal oleate.	Oleic Acid	106-112	cholecystokinin	Canis lupus familiaris	79-82	
6305210-6	1983	Neurotensin, like oleate, potentiated the action of secretin and CCK on pancreatic bicarbonate and had additive effects on protein response to these secretagogues.	Oleic Acid	18-24	cholecystokinin	Canis lupus familiaris	65-68	
2870644-4	1986	Perfusion of the intact intestine with graded amounts of oleate (0.5-16 mmol/h) produced dose-dependent increments in plasma CCK and pancreatic protein similar to those obtained with intravenous infusion of graded doses of CCK (0.85-255 pmol X kg-1 X h-1).	Oleic Acid	57-63	cholecystokinin	Canis lupus familiaris	125-128	
2870644-4	1986	Perfusion of the intact intestine with graded amounts of oleate (0.5-16 mmol/h) produced dose-dependent increments in plasma CCK and pancreatic protein similar to those obtained with intravenous infusion of graded doses of CCK (0.85-255 pmol X kg-1 X h-1).	Oleic Acid	57-63	cholecystokinin	Canis lupus familiaris	223-226	
1130517-7	1975	The decreased pancreatic response to intestinally perfused oleate and tryptophan seen after extragastric vagotomy could be caused either by interruption of reflex paths between gut and pancreas or by interference with CCK release.	Oleic Acid	59-65	cholecystokinin	Canis lupus familiaris	218-221