PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31583906-4	2022	Cells treated with BCP, in a dose-dependent manner, exhibited morphological changes, showed lower cell growth, underwent apoptosis and lost the ability to metastasis through the suppression of NF-k B via PI3K/AKT signalling pathway.	caryophyllene	19-22	AKT serine/threonine kinase 1	Homo sapiens	209-212	
31583906-5	2022	These results elucidate that the inhibition of NF-k B and PI3K/AKT is one of the most important mechanism by which BCP suppresses cancer cell proliferation and enhances apoptosis.	caryophyllene	115-118	AKT serine/threonine kinase 1	Homo sapiens	63-66	
34318533-0	2021	beta-Caryophyllene induces apoptosis and inhibits cell proliferation by deregulation of STAT-3/mTOR/AKT signaling in human bladder cancer cells: An in vitro study.	caryophyllene	0-18	AKT serine/threonine kinase 1	Homo sapiens	100-103	
34318533-1	2021	The current study aimed to explore the antitumor effect of beta-caryophyllene (BCP) on two different cell lines of T24 and 5637 human bladder cancer (BC) cells and its potential molecular mechanisms in inhibition of STAT-3/mTOR/AKT signaling pathways and the inductive process of apoptosis mechanism.	caryophyllene	59-77	AKT serine/threonine kinase 1	Homo sapiens	228-231	
34318533-1	2021	The current study aimed to explore the antitumor effect of beta-caryophyllene (BCP) on two different cell lines of T24 and 5637 human bladder cancer (BC) cells and its potential molecular mechanisms in inhibition of STAT-3/mTOR/AKT signaling pathways and the inductive process of apoptosis mechanism.	caryophyllene	79-82	AKT serine/threonine kinase 1	Homo sapiens	228-231