PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10452119-6	1999	When the [Ca2+]i store was depleted with ionomycin in the presence of egtazic acid (EGTA), the increase in pHi induced by thrombin was inhibited.	Ionomycin	41-50	glucose-6-phosphate isomerase	Oryctolagus cuniculus	107-110	
9156340-7	1997	A concentration-response relationship involving changes in pHi was easily demonstrated in CE cells after treatment with ionomycin, a calcium ionopore.	Ionomycin	120-129	glucose-6-phosphate isomerase	Oryctolagus cuniculus	59-62	
8835317-8	1996	Treatment of CE cells with the calcium ionophores, ionomycin and 4-bromo A23187, increased [Ca++]i, but produced a biphasic change in pHi.	Ionomycin	51-60	glucose-6-phosphate isomerase	Oryctolagus cuniculus	134-137	
8835317-10	1996	When [Ca++]i was decreased by treating the cells with 5 mM EGTA and 20 microM ionomycin, pHi decreased by 0.35 +/- 0.02 units.	Ionomycin	78-87	glucose-6-phosphate isomerase	Oryctolagus cuniculus	89-92	
7893996-2	1994	pHi was assessed with 2",7"-bis-(2-carboxyethyl)-5-carboxyfluorescein during proximal tubule injury produced by simple ATP depletion with the electron transport inhibitor antimycin or by large increases of cytosolic free Ca2+ induced by treatment with the calcium ionophore ionomycin, alone and in combination with antimycin.	Ionomycin	274-283	glucose-6-phosphate isomerase	Oryctolagus cuniculus	0-3