PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25889730-4	2015	Previously, we showed, using 7,12-dimethylbenz [a] anthracene (DMBA)-treated haploinsufficient PPARgamma mice, that PPARgamma suppresses breast tumour progression; however, the PPARgamma expressing cell types and mechanisms involved remain to be clarified.	7,12-dimethylbenz	29-46	peroxisome proliferator activated receptor gamma	Mus musculus	116-125	
25889730-4	2015	Previously, we showed, using 7,12-dimethylbenz [a] anthracene (DMBA)-treated haploinsufficient PPARgamma mice, that PPARgamma suppresses breast tumour progression; however, the PPARgamma expressing cell types and mechanisms involved remain to be clarified.	7,12-dimethylbenz	29-46	peroxisome proliferator activated receptor gamma	Mus musculus	116-125	
24481326-5	2014	Treatment with the PPARgamma agonist efatutazone in a mouse with Brca1 and p53 deficiency and 7,12-dimethylbenz(a)anthracene exposure in combination with an activated myristoylated form of AKT1 also induce ER+ mammary cancer.	7,12-dimethylbenz	94-111	peroxisome proliferator activated receptor gamma	Mus musculus	19-28	
15864802-3	2005	The recent finding that mice lacking one allele of the PPARgamma gene were significantly more susceptible to 7,12-dimethylbenz[a]anthracene (DMBA)-induced skin carcinogenesis compared to wild-type mice highlights mouse skin as another potential organ in which PPARgamma ligands may be effective as chemopreventive agents.	7,12-dimethylbenz	109-126	peroxisome proliferator activated receptor gamma	Mus musculus	55-64	
23934545-2	2014	Using peroxisome proliferator-activated receptor (PPAR)gamma((+/-)) mice, we showed normal expression of PPARgamma was critical to stop 7,12-dimethylbenz[a]anthracene (DMBA)-induced breast tumorigenesis.	7,12-dimethylbenz	136-153	peroxisome proliferator activated receptor gamma	Mus musculus	105-114